نتایج جستجو برای: prion protein

تعداد نتایج: 1238350  

Journal: :journal of physical & theoretical chemistry 2011
s. mansouri

the molecular dynamics (md) simulation method is used to assess structural details for humanprion protein (hereafter prpn) and its asp178 asn mutant (hereafter prpm) which causes fatalfamilial insomnia disease. the results reveal that the flexibility and instability increase in prpmcould be related to specific amino acids exposed to the solvent. solvation free energy of prpm is 20kjmot1nni2 mor...

Journal: :World Journal of Biology Pharmacy and Health Sciences 2022

If the attacking force has a vaccine before attack, will inflict crippling damage on enemy. Thus, virus emerges as potential danger since they can be used in development of such weapons. The reported neurotropic strain influenza A (IAV) induced conversion normal prion protein (PrPC) into infectious (PrPSc) well formation prions bring out type prion/virus unprecedented pathogens that cause fatal...

Journal: :Acta biochimica et biophysica Sinica 2013
Zhen Yuan Deming Zhao Lifeng Yang

Rabbits have low susceptibility to prion infection. Studies on prion protein (PrP) from animal species of different susceptibility to prion diseases identified key amino acid residues, specific motif, and special features in rabbit prion protein (RaPrP(C)) that contribute to the stability of rabbit PrP(C) and low susceptibility to prion infection. However, there is no evidence showing that rabb...

2010
Wei Hu Stefan Nessler Bernhard Hemmer Todd N. Eagar Lawrence P. Kane S. Rutger Leliveld Andreas Müller-Schiffmann Anne R. Gocke Amy Lovett-Racke Li-Hong Ben Rehana Z. Hussain Andreas Breil Jeffrey L. Elliott Krishna Puttaparthi Petra D. Cravens Mahendra P. Singh Benjamin Petsch Lothar Stitz Michael K. Racke Carsten Korth Olaf Stüve

The primary biological function of the endogenous cellular prion protein has remained unclear. We investigated its biological function in the generation of cellular immune responses using cellular prion protein gene-specific small interfering ribonucleic acid in vivo and in vitro. Our results were confirmed by blocking cellular prion protein with monovalent antibodies and by using cellular prio...

2012
Nicole T. Watt David R. Taylor Talitha L. Kerrigan Heledd H. Griffiths Jo V. Rushworth Isobel J. Whitehouse Nigel M. Hooper

Zinc is released into the synaptic cleft upon exocytotic stimuli, although the mechanism for its reuptake into neurons is unresolved. Here we show that the cellular prion protein enhances the uptake of zinc into neuronal cells. This prion-protein-mediated zinc influx requires the octapeptide repeats and amino-terminal polybasic region in the prion protein, but not its endocytosis. Selective ant...

2015
Pamela J. Skinner Hyeon O. Kim Damani Bryant Nikilyn J. Kinzel Cavan Reilly Suzette A. Priola Anne E. Ward Patricia A. Goodman Katherine Olson Davis M. Seelig Ina Maja Vorberg

Prion diseases such as Creutzfeldt-Jakob disease in humans, bovine spongiform encephalopathy in cattle, and scrapie in sheep are fatal neurodegenerative diseases for which there is no effective treatment. The pathology of these diseases involves the conversion of a protease sensitive form of the cellular prion protein (PrPC) into a protease resistant infectious form (PrPsc or PrPres). Both in v...

2016
Ji-Hong Moon Ju-Hee Lee Uddin MD Nazim You-Jin Lee Jae-Won Seol Seong-Kug Eo John-hwa Lee Sang-Youel Park

An unusual molecular structure of the prion protein, PrPsc is found only in mammals with transmissible prion diseases. Prion protein stands for either the infectious pathogen itself or a main component of it. Recent studies suggest that autophagy is one of the major functions that keep cells alive and has a protective effect against the neurodegeneration. In this study, we investigated that the...

Journal: :genetics in the 3rd millennium 0
امید آریانی omid aryani special medical center, tehran, iran مسعود هوشمند massoud houshmand فرهاد عصار زادگان farhad assarzadegan

protein aggregation is thought to be the pathological driving force responsible for neurodegenerative disorders such as alzheimer’s, parkinson’s, huntington’s, frontotemporal dementia, dementia with lewy bodies and prion diseases, however, it is not yet clear whether, or to what extent, the misfolded proteins are the cause of the diseases rather than the consequences. the aggregated proteins th...

Journal: :The Journal of biological chemistry 2002
Yaping Gu Hisashi Fujioka Ravi Shankar Mishra Ruliang Li Neena Singh

In infectious and familial prion disorders, neurodegeneration is often seen without obvious deposits of the scrapie prion protein (PrP(Sc)), the principal cause of neuronal death in prion disorders. In such cases, neurotoxicity must be mediated by alternative pathways of cell death. One such pathway is through a transmembrane form of PrP. We have investigated the relationship between intracellu...

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