نتایج جستجو برای: airway inflammation

تعداد نتایج: 243898  

Journal: :Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology 2006
S Furusho S Myou M Fujimura T Kita M Yasui K Kasahara S Nakao K Takehara S Sato

BACKGROUND Adhesion molecules such as intercellular adhesion molecule-1 (ICAM-1) are thought to contribute to the airway inflammation and airway hyper-responsiveness (AHR) of allergic asthma. Some differences from allergic asthma have been noted, including airway neutrophilia, and the involvement of ICAM-1 in toluene diisocyanate (TDI) asthma is currently unclear. OBJECTIVE We utilized mice l...

2014
Jung Yeon Shim

Asthma comprises a heterogeneous group of disorders characterized by airway inflammation, airway obstruction, and airway hyperresponsiveness (AHR). Airway inflammation, which induces AHR and recurrence of asthma, is the main pathophysiology of asthma. The fractional exhaled nitric oxide (FeNO) level is a noninvasive, reproducible measurement of eosinophilic airway inflammation that is easy to p...

Journal: :Journal of Allergy and Clinical Immunology 2019

Journal: :Nature Reviews Immunology 2013

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2017
Long P Nguyen Nour A Al-Sawalha Sergio Parra Indira Pokkunuri Ozozoma Omoluabi Adedoyin A Okulate Elizabeth Windham Li Matthew Hazen Jose M Gonzalez-Granado Craig J Daly John C McGrath Michael J Tuvim Brian J Knoll Burton F Dickey Richard A Bond

The mostly widely used bronchodilators in asthma therapy are β2-adrenoreceptor (β2AR) agonists, but their chronic use causes paradoxical adverse effects. We have previously determined that β2AR activation is required for expression of the asthma phenotype in mice, but the cell types involved are unknown. We now demonstrate that β2AR signaling in the airway epithelium is sufficient to mediate ke...

2015
Hoang Kim Tu Trinh Hae-Sim Park Yoo Seob Shin Dong-Hyeon Suh Jing-Nan Liu

Background and objective Cysteinyl leukotrienes (LTs) contribute to airway inflammation by interacting with type 1 and 2 cysteinyl leukotriene receptors as well as not clearly known type 3 leukotriene receptors. In contrast to LTC4 and LTD4, LTE4 is weak agonist at type 1 and 2 cysteinyl leukotriene receptors. Although LTE4 plays a key role in airway inflammation, our understanding of its poten...

2014
Matthew J Gold Michael R Hughes Frann Antignano Colby Zaph Kelly M McNagny

Background The PI3K pathway is a potent mediator of several functions associated with asthma pathogenesis, including supporting leukocyte survival, activation, migration and cytokine release. Proper negative regulation of this pathway is integral in order to restrict overactive immune responses. Negative regulation of PI3K is predominantly controlled by the lipid phosphatases PTEN and SHIP-1. I...

2016
Charity G. Duran Allison J. Burbank Katherine H. Mills Heather R. Duckworth Maria M. Aleman Matthew J. Kesic David B. Peden Yinghao Pan Haibo Zhou Michelle L. Hernandez

UNLABELLED Sulforaphane (SFN), a naturally occurring isothiocyanate found in cruciferous vegetables, is implicated as a possible therapy for airway inflammation via induction of the transcription factor NF-E2-related factor 2 (NRF2). In this proof-of-concept clinical study, we show that supplementation of SFN with broccoli sprout homogenate in healthy human subjects did not induce expression of...

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