نتایج جستجو برای: ctg repeat expansion

تعداد نتایج: 212195  

Journal: :genetics in the 3rd millennium 0
محمد حسین صنعتی mohammad hossein sanati molecular genetic diagnosis lab, special medical center, tehran, iran. امید آریانی omid ariani مسعود هوشمند masoud houshmand

myotonic dystrophy (dm), one of the most common forms of muscular dystrophy, is an inherited disorder of the muscles and other body systems. dm is a progressive genetic disorder with a triplet repeat autosomal mutation that affects an estimated 1 in 8000 people. myotonic dystrophy type 1, also known as steinerts disease, and type 2 caused by mutation in dmpk and cnbp genes, respectively. in eac...

Ali Fasihi Maryam Godarziyan Morteza Hashemzadeh-Chaleshtori Shahin Ramazi

Background & Aims: Nearly 30 hereditary disorders in humans result from an increase in the number of copies of simple repeats in genomic DNA, including fragile X syndrome, myotonic dystrophy, Huntington’s disease, and Friedreich’s ataxia. One the most frequently occurring types of mutation is trinucleotide repeat expansion. The present study was conducted with the aim of investigating the cause...

Journal: :PLoS ONE 2008
Warunee Dansithong Cordula M. Wolf Partha Sarkar Sharan Paul Andy Chiang Ian Holt Glenn E. Morris Dorothy Branco Megan C. Sherwood Lucio Comai Charles I. Berul Sita Reddy

The genetic basis of myotonic dystrophy type I (DM1) is the expansion of a CTG tract located in the 3' untranslated region of DMPK. Expression of mutant RNAs encoding expanded CUG repeats plays a central role in the development of cardiac disease in DM1. Expanded CUG tracts form both nuclear and cytoplasmic aggregates, yet the relative significance of such aggregates in eliciting DM1 pathology ...

Journal: :The American Journal of Human Genetics 1997

Journal: :Human molecular genetics 2012
Fernando Morales Jillian M Couto Catherine F Higham Grant Hogg Patricia Cuenca Claudia Braida Richard H Wilson Berit Adam Gerardo del Valle Roberto Brian Mauricio Sittenfeld Tetsuo Ashizawa Alison Wilcox Douglas E Wilcox Darren G Monckton

Deciphering the contribution of genetic instability in somatic cells is critical to our understanding of many human disorders. Myotonic dystrophy type 1 (DM1) is one such disorder that is caused by the expansion of a CTG repeat that shows extremely high levels of somatic instability. This somatic instability has compromised attempts to measure intergenerational repeat dynamics and infer genotyp...

Journal: :The Journal of Cell Biology 1995
K L Taneja M McCurrach M Schalling D Housman R H Singer

We have analyzed the intracellular localization of transcripts from the myotonin protein kinase (Mt-PK) gene in fibroblasts and muscle biopsies from myotonic dystrophy patients and normal controls. In affected individuals, a trinucleotide expansion in the gene results in the phenotype, the severity of which is proportional to the repeat length. A fluorochrome-conjugated probe (10 repeats of CAG...

Journal: :Current opinion in neurology 2002
Ami Mankodi Charles A Thornton

PURPOSE OF REVIEW To highlight recent advances in understanding the clinical manifestations and molecular genetics of myotonic syndromes, with particular emphasis on the myotonic dystrophies. RECENT FINDINGS Myotonic syndromes include the non-dystrophic myotonias, caused by mutations in genes encoding the chloride or sodium channels that are specific to skeletal muscle, and the myotonic dystr...

Journal: :Nucleic acids research 2001
Majid Fardaei Ken Larkin J. David Brook Marion G. Hamshere

Myotonic dystrophy (DM1) is the most common form of adult muscular dystrophy and is inherited as an autosomal dominant trait. The genetic basis of DM1 is the expansion of a CTG repeat in the 3' untranslated region of a protein kinase gene (DMPK). The molecular mechanism by which this expanded repeat produces the pathophysiology of DM1 remains unknown. Transcripts from the expanded allele accumu...

نمودار تعداد نتایج جستجو در هر سال

با کلیک روی نمودار نتایج را به سال انتشار فیلتر کنید