نتایج جستجو برای: fgfr3

تعداد نتایج: 1106  

Journal: :Journal of orthopaedic research : official publication of the Orthopaedic Research Society 2011
Arjun Sebastian Takehiko Matsushita Aya Kawanami Susan Mackem Gary E Landreth Shunichi Murakami

Activating mutations in FGFR3 cause the most common forms of human dwarfism: achondroplasia and thanatophoric dysplasia. In mouse models of achondroplasia, recent studies have implicated the ERK MAPK pathway, a pathway activated by FGFR3, in creating reduced bone growth. Our recent studies have indicated that increased Fgfr3 and ERK MAPK signaling in chondrocytes also causes premature synchondr...

Journal: :Mechanisms of Development 2000
Jennifer Walshe Ivor Mason

Studies involving chick embryos have implicated FGFs in neural induction and patterning as well as in other developmental events. Detailed analyses of FGF receptor expression at early stages of neural development have not been reported for the chick embryo and are incomplete for other vertebrate classes. Here we show the expression patterns of three FGF receptors, (FGFR1, FGFR2 and FGFR3) in em...

Journal: :Blood 1998
G L Vanham G Penne C Vereecken J Vingerhoets L Kestens

involves the fibroblast growth-factor receptor 3 gene. Blood 10:4062, 1997 9. Tavormina PL, Shiang R, Thompson LM, Zhu YZ, Wilkin DJ, Lachman RS, Wilcox WR, Rimoin DL, Cohn DH, Wasmuth JJ: Thanatophoric dysplasia (types I and II) caused by distinct mutations in fibroblast growth factor receptor 3. Nat Genet 9:321, 1995 10. Caligaris-Cappio F, Bergui L, Gregoretti MG, Gaidano G, Gaboli M, Schena...

Journal: :The Journal of clinical investigation 2009
Anna Mandinova Vihren Kolev Victor Neel Bing Hu Wesley Stonely Jocelyn Lieb Xunwei Wu Claudia Colli Rong Han Michael J Pazin Paola Ostano Reinhard Dummer Janice L Brissette G Paolo Dotto

Seborrheic keratoses (SKs) are common, benign epithelial tumors of the skin that do not, or very rarely, progress into malignancy, for reasons that are not understood. We investigated this by gene expression profiling of human SKs and cutaneous squamous cell carcinomas (SCCs) and found that several genes previously connected with keratinocyte tumor development were similarly modulated in SKs an...

Journal: :Carcinogenesis 2005
Hervé Wallerand Ashraf A Bakkar Sixtina Gil Diez de Medina Jean-Claude Pairon Yu-Ching Yang Dimitri Vordos Hugues Bittard Sylvie Fauconnet Jean-Claude Kouyoumdjian Marie-Claude Jaurand Zuo-Feng Zhang François Radvanyi Jean-Paul Thiery Dominique K Chopin

Smoking is a major risk factor for urothelial cell carcinoma of the bladder (UCC). Mutations in the FGFR3 and TP53 genes have been shown to define two distinct pathways in superficial papillary and invasive UCC disease, respectively. We investigated the relationship between smoking and these mutations by means of denaturing high performance liquid chromatography and sequencing for 110 primary U...

Journal: :Human molecular genetics 2006
G Valverde-Franco J S Binette W Li H Wang S Chai F Laflamme N Tran-Khanh E Quenneville T Meijers A R Poole J S Mort M D Buschmann J E Henderson

Fibroblast growth factor (FGF) receptor 3 has been identified as a key regulator of endochondral bone development and of post-natal bone metabolism through its action on growth plate chondrocytes and osteoblasts, respectively. It has also been shown to promote chondrogenesis and cartilage production by cultured pre-chondrogenic cells in response to FGF18. In the current studies, we show that th...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2014
Marzia Capelletti Michael E Dodge Dalia Ercan Peter S Hammerman Seung-Il Park Jhingook Kim Hidefumi Sasaki David M Jablons Doron Lipson Lauren Young Phil J Stephens Vincent A Miller Neal I Lindeman Kiara J Munir William G Richards Pasi A Jänne

PURPOSE Targetable oncogenic alterations are detected more commonly in patients with non-small cell lung cancer (NSCLC) who never smoked cigarettes. For such patients, specific kinase inhibitors have emerged as effective clinical treatments. However, the currently known oncogenic alterations do not account for all never smokers who develop NSCLC. We sought to identify additional oncogenic alter...

2015
Baolai Zhang Shuhong Dong Ruiming Zhu Chunyan Hu Jing Hou Yan Li Qian Zhao Xue Shao Qian Bu Hongyu Li Yongjie Wu Xiaobo Cen Yinglan Zhao

Protein arginine methyltransferases (PRMTs) plays critical roles in cancer. PRMT5 has been implicated in several types of tumors. However, the role of PRMT5 in cancer development remains to be fully elucidated. Here, we provide evidence that PRMT5 is overexpressed in colorectal cancer (CRC) cells and patient-derived primary tumors, correlated with increased cell growth and decreased overall pat...

Journal: :The Journal of clinical investigation 2013
Brittany C Parker Matti J Annala David E Cogdell Kirsi J Granberg Yan Sun Ping Ji Xia Li Joy Gumin Hong Zheng Limei Hu Olli Yli-Harja Hannu Haapasalo Tapio Visakorpi Xiuping Liu Chang-Gong Liu Raymond Sawaya Gregory N Fuller Kexin Chen Frederick F Lang Matti Nykter Wei Zhang

Fusion genes are chromosomal aberrations that are found in many cancers and can be used as prognostic markers and drug targets in clinical practice. Fusions can lead to production of oncogenic fusion proteins or to enhanced expression of oncogenes. Several recent studies have reported that some fusion genes can escape microRNA regulation via 3'-untranslated region (3'-UTR) deletion. We performe...

2016
Xiaotian Yuan Cheng Liu Kun Wang Li Liu Tiantian Liu Nan Ge Feng Kong Liu Yang Magnus Björkholm Yidong Fan Shengtian Zhao Dawei Xu

Urothelial cell carcinoma (UCC) includes urothelial bladder carcinoma (UBC), renal pelvic carcinoma (RPC) and ureter carcinoma (UC), and its incidence varies dependent on geographical areas and tumor locations, which indicates different oncogenic mechanisms and/or different genetic susceptibility/environment exposure. The activating mutations of the fibroblast growth factor receptor 3 (FGFR3) g...

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