نتایج جستجو برای: flt3 tyrosine kinase

تعداد نتایج: 262799  

2014
Caroline Benedicte Nitter Engen Line Wergeland Jørn Skavland Bjørn Tore Gjertsen Celalettin Ustun

Internal tandem duplications (ITDs) of the gene encoding the Fms-Like Tyrosine kinase-3 (FLT3) receptor are present in approximately 25% of patients with acute myeloid leukemia (AML). The mutation is associated with poor prognosis, and the aberrant protein product has been hypothesized as an attractive therapeutic target. Various tyrosine kinase inhibitors (TKIs) have been developed targeting F...

Journal: :Blood 2004
Patrick Brown Soheil Meshinchi Mark Levis Todd A Alonzo Robert Gerbing Beverly Lange Robert Arceci Donald Small

Pediatric acute myelogenous leukemia (AML) has a poor prognosis, and novel therapies are needed. The FLT3 tyrosine kinase represents a promising target in pediatric AML. FLT3 is constitutively activated either by an internal tandem duplication (ITD) or by a point mutation (PM) in 17% to 24% of pediatric AML cases. Autocrine stimulation of wild-type (WT) FLT3 by coexpressed FLT3 ligand (FL) occu...

Journal: :Blood 2008
Ulrike Bacher Claudia Haferlach Wolfgang Kern Torsten Haferlach Susanne Schnittger

We characterized the mutational status of the FLT3 tyrosine kinase domain (FLT3-TLD) in 3082 patients with newly diagnosed AML. FLT3-TKD mutations were detected in 147 of 3082 (4.8%) patients. Similar to the FLT3 juxtamembrane domain mutations (FLT3-LM), there was a high correlation of FLT3-TKD mutations with normal karyotype (88 of 1472; 6.0%). FLT3-TKD mutations were most frequent in the AML ...

Journal: :Blood 2002
Christian Thiede Christine Steudel Brigitte Mohr Markus Schaich Ulrike Schäkel Uwe Platzbecker Martin Wermke Martin Bornhäuser Markus Ritter Andreas Neubauer Gerhard Ehninger Thomas Illmer

Constitutive activation of the FLT3 receptor tyrosine kinase, either by internal tandem duplication (ITD) of the juxtamembrane region or by point mutations in the second tyrosine kinase domain (TKD), has been described in patients with acute myelogenous leukemia (AML). We analyzed the prevalence and the potential prognostic impact of FLT3 mutations in 979 AML patients. Results were correlated w...

Journal: :Journal of the National Comprehensive Cancer Network : JNCCN 2015
Smith Giri Shadi Hamdeh Vijaya Raj Bhatt James K Schwarz

Old age (≤65 years), relapsed or refractory disease, and the presence of FMS-like receptor tyrosine kinase-3 (FLT3) internal tandem duplication (ITD) mutation are poor prognostic factors in acute myeloid leukemia (AML). FLT3 inhibitors such as sorafenib have been shown to have a potential role in treating relapsed or refractory AML with FLT3 mutations. In the present report, the use of sorafeni...

Journal: :Asian Pacific journal of cancer prevention : APJCP 2010
Bircan Hizli Karabacak Fatih Erbey Ibrahim Bayram Sema Yilmaz Can Acipayam Yurdanur Kilinç Atila Tanyeli

INTRODUCTION In recent years, Fms-like tyrosine kinase (FLT) 3 has been the subject of several studies as a prognostic marker. OBJECTIVE In this study, the presence of FLT3 mutations in childhood acute leukemias patients and their association with prognosis were investigated. MATERIALS AND METHODS A total of 120 patients, 80 with acute lymphoblastic leukemia (ALL) and 40 with acute myelobla...

2017
Bradley Stockard Joy Guingab Timothy Garrett Stanley Pounds Jeffrey Rubnitz Jatinder Lamba

BACKGROUND/AIMS: Acute myeloid leukemia (AML) is a clinically challenging disease with high interpatient variability in response to chemotherapy. Despite continuing advances in treatment options, current 5-year survival rates for pediatric AML are suboptimal at ~60%. Variability in treatment response and survival outcomes are due in part to the heterogeneous nature of AML, with many genetic les...

Journal: :Blood 2010
Brunangelo Falini Katja Macijewski Tamara Weiss Ulrike Bacher Susanne Schnittger Wolfgang Kern Alexander Kohlmann Hans-Ulrich Klein Marco Vignetti Alfonso Piciocchi Paola Fazi Maria Paola Martelli Antonella Vitale Stefano Pileri Miriam Miesner Antonella Santucci Claudia Haferlach Franco Mandelli Torsten Haferlach

NPM1-mutated acute myeloid leukemia (AML) is a provisional entity in the 2008 World Health Organization (WHO) classification of myeloid neoplasms. The significance of multilineage dysplasia (MLD) in NPM1-mutated AML is unclear. Thus, in the 2008 WHO classification, NPM1-mutated AML with MLD is classified as AML with myelodysplasia (MD)-related changes (MRCs). We evaluated morphologically 318 NP...

Journal: :Clinical lymphoma, myeloma & leukemia 2015
Syed M Kazmi Naveen Pemmaraju Keyur P Patel Philip R Cohen Naval Daver Kathy M Tran Farhad Ravandi Madeleine Duvic Guillermo Garcia-Manero Sherry Pierce Aziz Nazha Gautam Borthakur Hagop Kantarjian Jorge Cortes

INTRODUCTION Sweet syndrome (SS) is associated with hematologic malignancies including acute myeloid leukemia (AML). PATIENTS AND METHODS Records of patients with AML treated at our institution were reviewed to identify those with SS. Patient characteristics, laboratory values, and cytogenetic and molecular abnormalities were retrospectively reviewed. RESULTS We identified 21 of 2178 (1%) A...

نمودار تعداد نتایج جستجو در هر سال

با کلیک روی نمودار نتایج را به سال انتشار فیلتر کنید