نتایج جستجو برای: homozygosity mapping

تعداد نتایج: 200921  

Journal: :Human heredity 2012
Emmanuelle Génin Mourad Sahbatou Steven Gazal Marie-Claude Babron Hervé Perdry Anne-Louise Leutenegger

To detect fully penetrant rare recessive variants that could constitute Mendelian subentities of complex diseases, we propose a novel strategy, the HBD-GWAS strategy, which can be applied to genome-wide association study (GWAS) data. This strategy first involves the identification of inbred individuals among cases using the genome-wide SNP data and then focuses on these inbred affected individu...

2011
Lu Zhang Wanling Yang Dingge Ying Stacey S Cherny Friedhelm Hildebrandt Pak Chung Sham Yu Lung Lau

Homozygosity mapping has played an important role in detecting recessive mutations using families of consanguineous marriages. However, detection of regions identical and homozygosity by descent (HBD) when family data are not available, or when relationships are unknown, is still a challenge. Making use of population data from high-density SNP genotyping may allow detection of regions HBD from ...

2012
Tobias Eisenberger Rima Slim Ahmad Mansour Markus Nauck Gudrun Nürnberg Peter Nürnberg Christian Decker Claudia Dafinger Inga Ebermann Carsten Bergmann Hanno Jörn Bolz

BACKGROUND Usher syndrome (USH) is an autosomal recessive genetically heterogeneous disorder with congenital sensorineural hearing impairment and retinitis pigmentosa (RP). We have identified a consanguineous Lebanese family with two affected members displaying progressive hearing loss, RP and cataracts, therefore clinically diagnosed as USH type 3 (USH3). Our study was aimed at the identificat...

2015
A Standing D Eleftheriou C Paisan-Ruiz D Rowcenzio Y Hong E Omoyinmi P Woo P Hawkins H Lachmann N Klein P Brogan

Patients and methods Three affected children in a Pakistani family suffered from a severe and unusual autoinflammatory syndrome, presenting in the first year of life with recurrent fevers, erythema nodosum-like rash, severe oromucocutaneous ulceration, systemic inflammation, and massively elevated serum IgD, without mutation in MVK. One of the affected children also suffered from multifocal ste...

2012
Marianne L. Voz Wouter Coppieters Isabelle Manfroid Ariane Baudhuin Virginie Von Berg Carole Charlier Dirk Meyer Wolfgang Driever Joseph A. Martial Bernard Peers

Forward genetics using zebrafish is a powerful tool for studying vertebrate development through large-scale mutagenesis. Nonetheless, the identification of the molecular lesion is still laborious and involves time-consuming genetic mapping. Here, we show that high-throughput sequencing of the whole zebrafish genome can directly locate the interval carrying the causative mutation and at the same...

2014
Marie Bernkopf Gerald Webersinke Chanakan Tongsook Chintan N. Koyani Muhammad A. Rafiq Muhammad Ayaz Doris Müller Christian Enzinger Muhammad Aslam Farooq Naeem Kurt Schmidt Karl Gruber Michael R. Speicher Ernst Malle Peter Macheroux Muhammad Ayub John B. Vincent Christian Windpassinger Hans-Christoph Duba

We describe the characterization of a gene for mild nonsyndromic autosomal recessive intellectual disability (ID) in two unrelated families, one from Austria, the other from Pakistan. Genome-wide single nucleotide polymorphism microarray analysis enabled us to define a region of homozygosity by descent on chromosome 17q25. Whole-exome sequencing and analysis of this region in an affected indivi...

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