نتایج جستجو برای: kruppel

تعداد نتایج: 729  

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2010
Won Jong Rhee Chih-Wen Ni Zhilan Zheng Kyunghwa Chang Hanjoong Jo Gang Bao

An important aspect of vascular biology is the identification of regulators of stress-sensitive genes that play critical roles in mediating inflammatory response. Here, we show that expression of HuR in human umbilical vein endothelial cells is regulated by shear stress and statin treatment; HuR, in turn, regulates other stress-sensitive genes such as Kruppel-like factor 2 (Klf2), endothelial n...

Journal: :Journal of applied physiology 2004
Juliann G Kiang Phillip D Bowman Brian W Wu Nyasa Hampton Andrew G Kiang Baiteng Zhao Yuang-Taung Juang James L Atkins George C Tsokos

The aim of this study was to determine whether hemorrhage affects the levels of a variety of stress-related proteins and whether changes can be inhibited by drugs reported to provide protection from ischemia and reperfusion injury. Male Swiss Webster mice were subjected to a 40% hemorrhage without resuscitation. Western blot analysis indicated that c-Jun (an AP-1 protein), Kruppel-like factor 6...

2013
Shaohui Hu Jun Wan Yijing Su Qifeng Song Yaxue Zeng Ha Nam Nguyen Jaehoon Shin Eric Cox Hee Sool Rho Crystal Woodard Shuli Xia Shuang Liu Huibin Lyu Guo-Li Ming Herschel Wade Hongjun Song Jiang Qian Heng Zhu

DNA methylation, especially CpG methylation at promoter regions, has been generally considered as a potent epigenetic modification that prohibits transcription factor (TF) recruitment, resulting in transcription suppression. Here, we used a protein microarray-based approach to systematically survey the entire human TF family and found numerous purified TFs with methylated CpG (mCpG)-dependent D...

Journal: :Cancer research 2008
Analisa DiFeo Lauren Feld Estefania Rodriguez Christine Wang David G Beer John A Martignetti Goutham Narla

Kruppel-like factor 6 (KLF6) is a tumor suppressor gene that is functionally inactivated in human cancer by loss of heterozygosity, somatic mutation, decreased expression, and increased alternative splicing into an oncogenic splice variant, KLF6-SV1. Here we show that increased expression of KLF6-SV1 is associated with decreased survival in patients with lung adenocarcinoma. In addition, KLF6-S...

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