نتایج جستجو برای: neurogenic inflammation

تعداد نتایج: 180208  

2014
Victor Meseguer Yeranddy A. Alpizar Enoch Luis Sendoa Tajada Bristol Denlinger Otto Fajardo Jan-Albert Manenschijn Carlos Fernández-Peña Arturo Talavera Tatiana Kichko Belén Navia Alicia Sánchez Rosa Señarís Peter Reeh María Teresa Pérez-García José Ramón López-López Thomas Voets Carlos Belmonte Karel Talavera Félix Viana

Gram-negative bacterial infections are accompanied by inflammation and somatic or visceral pain. These symptoms are generally attributed to sensitization of nociceptors by inflammatory mediators released by immune cells. Nociceptor sensitization during inflammation occurs through activation of the Toll-like receptor 4 (TLR4) signalling pathway by lipopolysaccharide (LPS), a toxic by-product of ...

Journal: :The Journal of neuroscience : the official journal of the Society for Neuroscience 2012
Michael Lattke Alexander Magnutzki Paul Walther Thomas Wirth Bernd Baumann

Hydrocephalus formation is a frequent complication of neuropathological insults associated with neuroinflammation. However, the mechanistic role of neuroinflammation in hydrocephalus development is unclear. We have investigated the function of the proinflammatory acting inhibitor of κB kinase (IKK)/nuclear factor κB (NF-κB) signaling system in neuroinflammatory processes and generated a novel m...

2015
Franca Orsini Daiana De Blasio Rosalia Zangari Elisa R. Zanier Maria-Grazia De Simoni

[This corrects the article on p. 380 in vol. 8, PMID: 25426028.].

2016
Luca Lorenzini Alessandro Giuliani Sandra Sivilia Vito Antonio Baldassarro Mercedes Fernandez Matteo Lotti Margotti Luciana Giardino Vania Fontani Salvatore Rinaldi Laura Calzà

The original version of this Article contained errors in the spelling of the authors Luca Lorenzini, Alessandro Giuliani, Sandra Sivilia, Vito Antonio Baldassarro, Mercedes Fernandez, Matteo Lotti Margotti, Luciana Giardino, Vania Fontani, Salvatore Rinaldi & Laura Calzà which were incorrectly given as Lorenzini Luca, Giuliani Alessandro, Sivilia Sandra, Baldassarro Vito Antonio, Fernandez Merc...

2010
Marcella Reale Talma Brenner Nigel H. Greig Nibaldo Inestrosa Diana Paleacu

1Department of Oncology and Neurosciences, ‘G. D’Annunzio’ University of Chieti-Pescara, Italy 2Department of Neurology, Hadassah Hebrew University Medical Center, P.O. Box 12000, Jerusalem 91120, Israel 3 Laboratory of Neurosciences, National Institute on Aging, Baltimore, MD 21224-6825, USA 4Centro de Envejecimiento y Regeneración, Centro de Regulación Celular y Patoloǵıa “Joaquı́n V. Luco,” I...

2012
Yukata Saito

In chronic prostatitis complaints of patients are diverse. Thus, often the disease is erroneously diagnosed as chronic cystitis, prostatomegaly, neurogenic cystitis, etc. Leader describes that chronic prostatitis is a stagnant uninfectious lesion resulting from an inflammation in the past. However, in many cases organisms are not detectable or, if detected, cannot be precisely related to the di...

Journal: :Frontiers in bioscience 2012
Xanthi Antoniou Tiziana Borsello

Among the numerous intracellular signalling pathways that control brain development and pathogenesis c-Jun N-terminal kinases have a leading role in the Central Nervous System. JNKs regulate a wide range of processes in brain development, plasticity, repair/regeneration, neuronal death and neuroinflammation. Indeed, accumulating evidence underline the potential of JNK targeted molecules towards...

2016
Michele Barry

Masters Candidate, Michele Barry, has successfully defended and made the required modifications to the text of the master's thesis for the M.S. during this Spring Semester 2016. THESIS COMMITTEE iv Acknowledgments I would like to express my sincere thanks to my advisor, Dr. Michael Vigorito, for his wisdom and guidance. His support has given me the opportunity to complete research in an area of...

2015
Chih-Wen Yang Jong-Ling Fuh

Exposure to anesthesia and surgery has been hypothesized to increase the risk of developing Alzheimer's disease (AD). While the exact pathogenesis of AD remains unknown, it potentially involves specific proteins (eg, amyloid beta and tau) and neuroinflammation. A growing body of preclinical evidence also suggests that anesthetic agents interact with the components that mediate AD neuropathology...

2017
Florence Thibaut

In psychiatric diseases such as mood disorders or schizophrenia, the inflammatory response system is activated. Microglia has gradually emerged as a key interface between stress-related signals and neuroimnune consequences of stress, with stressors leading to elevated microglial activity.

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