نتایج جستجو برای: neuroinflammation

تعداد نتایج: 6975  

Journal: :Journal of immunology 2006
Jozsef Karman Hamlet H Chu Dominic O Co Christine M Seroogy Matyas Sandor Zsuzsanna Fabry

Neuroinflammation often starts with the invasion of T lymphocytes into the CNS leading to recruitment of macrophages and amplification of inflammation. In this study, we show that dendritic cells (DCs) facilitate T-T cell help in the CNS and contribute to the amplification of local neuroinflammation. We adoptively transferred defined amounts of naive TCR-transgenic (TCR) recombination-activatin...

Journal: :The Lancet. Neurology 2015
Michael T Heneka Monica J Carson Joseph El Khoury Gary E Landreth Frederic Brosseron Douglas L Feinstein Andreas H Jacobs Tony Wyss-Coray Javier Vitorica Richard M Ransohoff Karl Herrup Sally A Frautschy Bente Finsen Guy C Brown Alexei Verkhratsky Koji Yamanaka Jari Koistinaho Eicke Latz Annett Halle Gabor C Petzold Terrence Town Dave Morgan Mari L Shinohara V Hugh Perry Clive Holmes Nicolas G Bazan David J Brooks Stéphane Hunot Bertrand Joseph Nikolaus Deigendesch Olga Garaschuk Erik Boddeke Charles A Dinarello John C Breitner Greg M Cole Douglas T Golenbock Markus P Kummer

Increasing evidence suggests that Alzheimer's disease pathogenesis is not restricted to the neuronal compartment, but includes strong interactions with immunological mechanisms in the brain. Misfolded and aggregated proteins bind to pattern recognition receptors on microglia and astroglia, and trigger an innate immune response characterised by release of inflammatory mediators, which contribute...

2014
Linda Block

Background

2017
Chiara Cerami Leonardo Iaccarino Daniela Perani

Neurodegeneration elicits neuroinflammatory responses to kill pathogens, clear debris and support tissue repair. Neuroinflammation is a dynamic biological response characterized by the recruitment of innate and adaptive immune system cells in the site of tissue damage. Resident microglia and infiltrating immune cells partake in the restoration of central nervous system homeostasis. Nevertheless...

2016
Chul Ju Hwang Hee Pom Lee Dong-Young Choi Heon Sang Jeong Tae Hoon Kim Tae Hyung Lee Young Min Kim Dae Bong Moon Sung Sik Park Sun Young Kim Ki-Wan Oh Dae Yeon Hwang Sang-Bae Han Hwa-Jeong Lee Jin Tae Hong

Neuroinflammation is implicated for dopaminergic neurodegeneration. Sulfur compounds extracted from garlic have been shown to have anti-inflammatory properties. Previously, we have investigated that thiacremonone, a sulfur compound isolated from garlic has anti-inflammatory effects on several inflammatory disease models. To investigate the protective effect of thiacremonone against 1-methyl-4-p...

2017

Alzheimer’s disease (AD) is the most common neurodegenerative disease characterized by neuronal loss due to amyloid beta (Aβ) aggregations, neurofibrillary tangles, and prominent neuroinflammation. Inflammatory processes in AD primarily occur in response to misfolded and aggregated proteins, or mislocalized nucleic acids and reactive microglia. Prolonged chronic neuroinflammation is thought to ...

2016
Marta Vicente-Rodríguez Rosalía Fernández-Calle Esther Gramage Carmen Pérez-García María P Ramos Gonzalo Herradón

Midkine (MK) is a cytokine that modulates amphetamine-induced striatal astrogliosis, suggesting a possible role of MK in neuroinflammation induced by amphetamine. To test this hypothesis, we studied astrogliosis and microglial response induced by amphetamine (10 mg/kg i.p. four times, every 2 h) in different brain areas of MK-/- mice and wild type (WT) mice. We found that amphetamine-induced mi...

2015
Megan E. Bosch Tammy Kielian

Lysosomal storage diseases (LSDs) include approximately 70 distinct disorders that collectively account for 14% of all inherited metabolic diseases. LSDs are caused by mutations in various enzymes/proteins that disrupt lysosomal function, which impairs macromolecule degradation following endosome-lysosome and phagosome-lysosome fusion and autophagy, ultimately disrupting cellular homeostasis. L...

Journal: :Neuroscience letters 2010
Holly M Brothers Yannick Marchalant Gary L Wenk

Caffeine is an antagonist at A1 and A2A adenosine receptors and epidemiological evidence suggests that caffeine consumption reduces the risk of Alzheimer's and Parkinson's diseases. Neuroinflammation plays a role in the etiology of these diseases and caffeine may provide protection through the modulation of inflammation. Adenosine has a known role in the propagation of inflammation and caffeine...

Journal: :The Journal of neuroscience : the official journal of the Society for Neuroscience 2012
Ofer Pasternak Carl-Fredrik Westin Sylvain Bouix Larry J Seidman Jill M Goldstein Tsung-Ung W Woo Tracey L Petryshen Raquelle I Mesholam-Gately Robert W McCarley Ron Kikinis Martha E Shenton Marek Kubicki

Diffusion MRI has been successful in identifying the existence of white matter abnormalities in schizophrenia in vivo. However, the role of these abnormalities in the etiology of schizophrenia is not well understood. Accumulating evidence from imaging, histological, genetic, and immunochemical studies support the involvement of axonal degeneration and neuroinflammation--ubiquitous components of...

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