نتایج جستجو برای: aptx gene

تعداد نتایج: 1141372  

2013
Guofeng Yue Jia Wei Xiaoping Qian Lixia Yu Zhengyun Zou Wenxian Guan Hao Wang Jie Shen Baorui Liu

OBJECTIVE The present study was designed to examine the anticancer effect of Traditional Chinese Medicine of polyphyllin I (PPI) and evodiamine (EVO) on freshly-removed gastric tumor tissues. METHODS Sixty freshly-removed gastric tumor tissues were collected. Their sensitivity to PPI, EVO, platinum (Pt), 5-FU, irinotecan (CPT-11) were determined by histoculture drug response assay (HDRA). Tho...

2017
Humera Manzoor Ihtisham Bukhari Muhammad Wajid Yuanwei Zhang Huan Zhang Norbert Brüggemann Christine Klein Qinghua Shi Sadaf Naz

Dear Editor, Ataxia with oculomotor apraxia type 1 (AOA1, MIM 208920) is an autosomal recessive disorder characterized by early-onset cerebellar ataxia, polyneuropathy, oculomotor apraxia, hypoalbuminemia, and hypercholesterolemia.1 It is caused by pathogenic variants of APTX, which encodes the aprataxin protein that is involved in DNA strand-break repair.1 We recruited a consanguineous family ...

Journal: :The FEBS journal 2013
Kimberly M Maize Carston R Wagner Barry C Finzel

The histidine triad proteins (HITs) constitute a large and ubiquitous superfamily of nucleotide hydrolases. The human histidine triad nucleotide-binding proteins (hHints) are a distinct class of HITs noted for their acyl-AMP hydrolase and phosphoramidase activity. The first high-resolution crystal structures of hHint2 with and without bound AMP are described. The differences between hHint2 and ...

Journal: :Mechanisms of ageing and development 2011
Keith W Caldecott Vilhelm A Bohr Peter J McKinnon

Ataxia telangiectasia (ATM), AT like disorder (MRE11), AOA1 (APTX) and AOA2 (SETX) in the UK – variability of the neurological, genetic and cellular phenotypes 19.05-20.00 Jan Hoeijmakers (Netherlands) The link between DNA damage, global and transcription coupled repair and neurodegeneration Mark O'Driscoll (UK) Defective genome stability and impaired neurogenesis in congenital human disorders ...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2010
Higinio Dopeso Silvia Mateo-Lozano Elena Elez Stefania Landolfi Francisco Javier Ramos Pascual Javier Hernández-Losa Rocco Mazzolini Paulo Rodrigues Sarah Bazzocco Maria Josep Carreras Eloy Espín Manel Armengol Andrew J Wilson John M Mariadason Santiago Ramon Y Cajal Josep Tabernero Simo Schwartz Diego Arango

PURPOSE Irinotecan (CPT11) treatment significantly improves the survival of colorectal cancer patients and is routinely used for the treatment of these patients, alone or in combination with other agents. However, only 20% to 30% of patients show an objective response to irinotecan, and there is great need for new molecular markers capable of identifying the subset of patients who are unlikely ...

Journal: :Current Biology 2007
David M. Wilson Mark P. Mattson

Ataxia oculomotor apraxia-1 is a neurological disorder that arises from mutations in the gene encoding the protein aprataxin. A recent study demonstrates that aprataxin is critical for the processing of obstructive DNA termini, suggesting a broader role for DNA single-strand break repair in neurodegenerative disease.

Journal: :Journal of neurological disorders & stroke 2013
Catarina M Quinzii Michio Hirano Ali Naini

In 2001, we described six patients with cerebellar ataxia and severe deficiency of coenzyme Q10 (CoQ10, ubiquinone) in skeletal muscle [1]. Within one year, we described 13 additional patients [2]; therefore, we suspected this was not a very rare syndrome. Twelve years after our original report, cerebellar ataxia and atrophy has emerged as the most common clinical presentation of CoQ10 deficien...

Journal: :Journal of clinical neurology 2016
Minwoo Lee Nan Young Kim Jin Young Huh Young Eun Kim Yun Joong Kim

Dear Editor, Ataxia with oculomotor apraxia type I (AOA1) is a recessively inherited ataxic disorder that is characterized clinically by the childhood onset of progressive cerebellar ataxia, oculomotor apraxia (OMA), and peripheral axonal sensorimotor neuropathy.1 Dystonia, chorea, and cognitive impairment are commonly associated symptoms, and hypoalbuminemia and hypercholesterolemia are often ...

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