نتایج جستجو برای: cd95l

تعداد نتایج: 278  

Journal: :Cancer research 2001
S T Eichhorst S Müerköster M A Weigand P H Krammer

The CD95/CD95 ligand (CD95L) system has been shown to mediate chemotherapeutic drug-induced apoptosis in vitro. However, the contribution of the CD95 pathway to drug-induced apoptosis is controversial. We have shown previously that 5-fluorouracil (5-FU) induces apoptosis in vitro via the activation of the CD95/CD95L system. To study the effects of the chemotherapeutic drug 5-FU and the contribu...

صیاد, آرزو, فراز, عاطفه, یاری, رضا,

سابقه و هدف: بیماری مولتیپل اسکلروزیس از رایج ترین بیماری ‌های التهابی سیستم اعصاب مرکزی است. در افراد بیمار، اختلال در مسیر آپوپتوز وجود دارد که این باعث می ‌شود سلول‌ های T بیش فعال، آپوپتوز نشده و وارد جریان خون شوند. در میان ژن ‌های مرتبط با بیماری مذکور، ژنی به نام CD95L وجود دارد که در مسیر آپوپتوز نقش ایفا می‌کند. مواد و روش ها: در این مطالعه از 30 نفر بیمار مبتلا به مولتیپل اسکلروزیس و ...

2017
William Putzbach Quan Q Gao Monal Patel Stijn van Dongen Ashley Haluck-Kangas Aishe A Sarshad Elizabeth T Bartom Kwang-Youn A Kim Denise M Scholtens Markus Hafner Jonathan C Zhao Andrea E Murmann Marcus E Peter

Over 80% of multiple-tested siRNAs and shRNAs targeting CD95 or CD95 ligand (CD95L) induce a form of cell death characterized by simultaneous activation of multiple cell death pathways preferentially killing transformed and cancer stem cells. We now show these si/shRNAs kill cancer cells through canonical RNAi by targeting the 3'UTR of critical survival genes in a unique form of off-target effe...

2017
Matthieu Le Gallo Amanda Poissonnier Patrick Blanco Patrick Legembre

Endothelial cells lining new blood vessels that develop during inflammatory disorders or cancers act as doors that either allow or block access to the tumor or inflamed organ. Recent data show that these endothelial cells in cancer tissues and inflamed tissues of lupus patients overexpress CD95L, the biological role of which is a subject of debate. The receptor CD95 (also named Fas or apoptosis...

2017
William Putzbach Quan Q. Gao Monal Patel Aishe A. Sarshad Abbas Hadji Stijn van Dongen Ashley Haluck-Kangas Elizabeth Bartom Austin Stults Abdul S. Qadir Kwang-Youn A. Kim Markus Hafner Jonathan C. Zhao Andrea E. Murmann Marcus E. Peter

>80% of a large number of siRNAs and shRNAs targeting CD95 or CD95 ligand (CD95L) induce a form of cell death that is characterized by the simultaneous activation of multiple death pathways and preferentially affects transformed and cancer stem cells. We now show that these si/shRNAs kill cancer cells through canonical RNAi by targeting the 3’UTR of critical survival genes in a unique form of o...

2017
Andrea E. Murmann Kaylin M. McMahon Ashley Haluck-Kangas Nandini Ravindran Monal Patel Calvin Y. Law Sonia Brockway Jian-Jun Wei C. Shad Thaxton Marcus E. Peter

The death receptor CD95/Fas can be activated by immune cells to kill cancer cells. shRNAs and siRNAs derived from CD95 or CD95 ligand (CD95L) are highly toxic to most cancer cells. We recently found that these sh/siRNAs kill cancer cells in the absence of the target by targeting the 3'UTRs of critical survival genes through canonical RNAi. We have named this unique form of off-target effect DIS...

Journal: :Blood 2007
Dirk Brenner Alexander Golks Mareike Becker Wolfgang Müller Christian R Frey Rostislav Novak Doron Melamed Friedemann Kiefer Peter H Krammer Rüdiger Arnold

Life and death of peripheral lymphocytes is strictly controlled to maintain physiologic levels of T and B cells. Activation-induced cell death (AICD) is one mechanism to delete superfluous lymphocytes by restimulation of their immunoreceptors and it depends partially on the CD95/CD95L system. Recently, we have shown that hematopoietic progenitor kinase 1 (HPK1) determines T-cell fate. While ful...

ژورنال: :مجله تازه های بیوتکنولوژی سلولی - مولکولی 0
عاطفه فراز atefeh faraz department of biology, faculty of sciences, islamic azad university, boroujerd branch, boroujerd, iranگروه زیست شناسی، دانشکده علوم، واحد بروجرد، دانشگاه آزاد اسلامی، بروجرد، ایران آرزو صیاد arezou sayad department of medical genetics, shahid beheshti university of medical sciences, tehran, iranگروه ژنتیک پزشکی، دانشکده پزشکی، دانشگاه شهید بهشتی، تهران، ایران رضا یاری reza yari department of biology, faculty of sciences, islamic azad university, boroujerd branch, boroujerd, iranگروه زیست شناسی، دانشکده علوم، واحد بروجرد، دانشگاه آزاد اسلامی، بروجرد، ایران

سابقه و هدف: بیماری مولتیپل اسکلروزیس از رایج ترین بیماری های التهابی سیستم اعصاب مرکزی است. در افراد بیمار، اختلال در مسیر آپوپتوز وجود دارد که این باعث می شود سلول های t بیش فعال، آپوپتوز نشده و وارد جریان خون شوند. در میان ژن های مرتبط با بیماری مذکور، ژنی به نام cd95l وجود دارد که در مسیر آپوپتوز نقش ایفا می کند. مواد و روش ها: در این مطالعه از 30 نفر بیمار مبتلا به مولتیپل اسکلروزیس و 30 ف...

Journal: :Blood 2002
Anne Janin Christophe Deschaumes Marjan Daneshpouy Jérôme Estaquier Juliette Micic-Polianski Premavathy Rajagopalan-Levasseur Khadija Akarid Nicolas Mounier Eliane Gluckman Gérard Socié Jean Claude Ameisen

Fas (CD95) is a death receptor involved in apoptosis induction on engagement by Fas ligand (CD95L). Although CD95L-mediated apoptosis has been proposed as a pathogenic mechanism in a wide range of diseases, including graft-versus-host disease, systemic CD95 engagement in mice by agonistic CD95-specific antibodies or by soluble multimeric CD95L (smCD95L), though lethal, has been reported to caus...

Journal: :Journal of immunology 2000
T Waku T Fujiwara J Shao T Itoshima T Murakami M Kataoka S Gomi J A Roth N Tanaka

Clinical trials of adenoviral p53 gene therapy provide the evidence that the bystander effect induced by the wild-type p53 gene transfer on adjacent tumor cells contributes to tumor progression; its mechanism, however, remains uncharacterized. We report in this work that injection of adenovirus expressing the human wild-type p53 gene (Ad5CMVp53) into established human colorectal tumors in nu/nu...

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