نتایج جستجو برای: kallmann syndrome

تعداد نتایج: 621960  

Journal: :AJNR. American journal of neuroradiology 1993
J R Knorr R L Ragland R S Brown N Gelber

PURPOSE To evaluate patients with known hypogonadotropic hypogonadism, some with known anosmia, for defective rhinocephalon development that resulted in olfactory tract abnormalities, an affliction known as Kallmann syndrome. METHODS Six patients who clinically had hypogonadotropic hypogonadism were examined by MR. Thin coronal images of the interior frontal region were used to determine pres...

Journal: :Brain : a journal of neurology 2013
Sheena Chew Ravikumar Balasubramanian Wai-Man Chan Peter B Kang Caroline Andrews Bryn D Webb Sarah E MacKinnon Darren T Oystreck Jessica Rankin Thomas O Crawford Michael Geraghty Scott L Pomeroy William F Crowley Ethylin Wang Jabs David G Hunter Patricia E Grant Elizabeth C Engle

Missense mutations in TUBB3, the gene that encodes the neuronal-specific protein β-tubulin isotype 3, can cause isolated or syndromic congenital fibrosis of the extraocular muscles, a form of complex congenital strabismus characterized by cranial nerve misguidance. One of the eight TUBB3 mutations reported to cause congenital fibrosis of the extraocular muscles, c.1228G>A results in a TUBB3 E41...

Journal: :Endocrine journal 1999
Y Izumi K Tatsumi S Okamoto A Hosokawa S Ueno H Fukui N Amino

Kallmann syndrome is defined by the association of hypogonadotropic hypogonadism and anosmia, for which three modes of transmission have been described: X-linked, autosomal recessive and autosomal dominant. The KAL1 gene, responsible for the X-linked form of the disease, has been isolated and its intron-exon organization determined. We report sequence analysis using PCR-direct sequencing method...

Journal: :Neurology India 2004
R Madan Vijay Sawlani Sushil Gupta R V Phadke

Kallmann syndrome (KS) is a neuronal migration disorder characterised by hypogonadotrophic hypogonadism and anosmia or hyposmia. Five patients with clinical findings suggestive of KS were evaluated with MRI. All patients had abnormalities of olfactory system. Olfactory bulbs were absent in all patients. Olfactory sulci were absent in 3 patients and hypoplastic in 2 patients. Anterior pituitary ...

Journal: :PLoS Genetics 2006
Catherine Dodé Luis Teixeira Jacqueline Levilliers Corinne Fouveaut Philippe Bouchard Marie-Laure Kottler James Lespinasse Anne Lienhardt-Roussie Michèle Mathieu Alexandre Moerman Graeme Morgan Arnaud Murat Jean-Edmont Toublanc Slawomir Wolczynski Marc Delpech Christine Petit Jacques Young Jean-Pierre Hardelin

Kallmann syndrome combines anosmia, related to defective olfactory bulb morphogenesis, and hypogonadism due to gonadotropin-releasing hormone deficiency. Loss-of-function mutations in KAL1 and FGFR1 underlie the X chromosome-linked form and an autosomal dominant form of the disease, respectively. Mutations in these genes, however, only account for approximately 20% of all Kallmann syndrome case...

Journal: :The Journal of Clinical Endocrinology & Metabolism 2013

Journal: :Frontiers in Endocrinology 2013

Journal: :Proceedings of the National Academy of Sciences of the United States of America 1989
A Ballabio B Bardoni R Carrozzo G Andria D Bick L Campbell B Hamel M A Ferguson-Smith G Gimelli M Fraccaro

Mendelian inherited disorders due to deletions of adjacent genes on a chromosome have been described as "contiguous gene syndromes." Short stature, chondrodysplasia punctata, mental retardation, steroid sulfatase deficiency, and Kallmann syndrome have been found as isolated entities or associated in various combinations in 27 patients with interstitial and terminal deletions involving the dista...

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