نتایج جستجو برای: mitochondriopathy

تعداد نتایج: 62  

Journal: :Cell reports 2016
Nadine Assmann Katja Dettmer Johann M B Simbuerger Carsten Broeker Nadine Nuernberger Kathrin Renner Holly Courtneidge Enriko D Klootwijk Axel Duerkop Andrew Hall Robert Kleta Peter J Oefner Markus Reichold Joerg Reinders

We recently reported an autosomal dominant form of renal Fanconi syndrome caused by a missense mutation in the third codon of the peroxisomal protein EHHADH. The mutation mistargets EHHADH to mitochondria, thereby impairing mitochondrial energy production and, consequently, reabsorption of electrolytes and low-molecular-weight nutrients in the proximal tubule. Here, we further elucidate the mol...

Journal: :Journal of neuropathology and experimental neurology 2006
Lee J Martin

The causes of the selective neurodegeneration in Parkinson disease (PD) and amyotrophic lateral sclerosis (ALS) are unknown and commonalities among these and other age-related neurodegenerative diseases continue to be sought. Morphologic, biochemical, molecular genetic, and cell/animal model studies suggest that mitochondria might be a convergence point for neurodegeneration. The functions and ...

Journal: :Human molecular genetics 1997
K Oexle A Zwirner

Cell and tissue damage in respiratory chain disorders have been related to increased production of reactive oxygen species (ROS). We measured telomere lengths in such disorders since ROS have also been implicated with telomere shortening. We investigated whole blood cell DNA of 14 patients with MELAS-related mitochondriopathy and two patients with the LHON-associated G11778A mutation of the mit...

2017
Paule Bénit Alice Pelhaître Elise Saunier Sylvie Bortoli Assetou Coulibaly Malgorzata Rak Manuel Schiff Guido Kroemer Massimo Zeviani Pierre Rustin

Mice with the hypomorphic AIF-Harlequin mutation exhibit a highly heterogeneous mitochondriopathy that mostly affects respiratory chain complex I, causing a cerebral pathology that resembles that found in patients with AIF loss-of-function mutations. Here we describe that the antidiabetic drug pioglitazone (PIO) can improve the phenotype of a mouse Harlequin (Hq) subgroup, presumably due to an ...

Journal: :Neuro endocrinology letters 2004
Rüdiger Lorenz

An initial report on the therapeutic application of delta 9-THC (THC) (Dronabinol, Marinol) in 8 children resp. adolescents suffering from the following conditions, is given: neurodegenerative disease, mitochondriopathy, posthypoxic state, epilepsy, posttraumatic reaction. THC effected reduced spasticity, improved dystonia, increased initiative (with low dose), increased interest in the surroun...

نمودار تعداد نتایج جستجو در هر سال

با کلیک روی نمودار نتایج را به سال انتشار فیلتر کنید