نتایج جستجو برای: proteinase 3 pr3

تعداد نتایج: 1820720  

Journal: :Arthritis Research 2000
Elisabeth Peen Williams Ralph C

Reactive antigenic epitopes on presumed autoantigens of biologic interest have been examined by many researchers. The central third complementarity-determining region (CDR3) residues of a human monoclonal anti-proteinase 3 (PR3) antibody contained many negatively charged aspartic acid residues, perhaps contributing to its reactivity with positively charged PR3 regions. Examination of four other...

Journal: :Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association 2009
Silvia M Uriarte Kenneth R McLeish Richard A Ward

BACKGROUND Anti-neutrophil cytoplasmic antibodies (ANCA) against proteinase 3 (PR3) are postulated to injure vascular endothelium by inducing cytokine-primed neutrophils to release proteolytic enzymes and generate reactive oxygen species. Anti-PR3 induce exocytosis, and since priming is associated with upregulation of plasma membrane proteins by exocytosis of intracellular granules, we tested t...

Journal: :Postgraduate medical journal 2000
S Lee J W Lawton

Thirty seven patients tested positive for antibody against proteinase 3 (anti-PR3) using either one of the two in-house ELISA methods at the Immunology Laboratory, University of Hong Kong. All except three were Chinese. Systemic vasculitis was diagnosed in 13 patients. However, the positive predictive value of anti-PR3 for Wegener's granulomatosus was low (22%). The commonest presenting feature...

2012
Mark A. Little Bahjat Al-Ani Shuyu Ren Hamad Al-Nuaimi Maurilo Leite Charles E. Alpers Caroline O. Savage Jeremy S. Duffield

Evidence is lacking for direct pathogenicity of human anti-proteinase-3 (PR3) antibodies in development of systemic vasculitis and granulomatosis with polyangiitis (GPA, Wegener's granulomatosis). Progress in study of these antibodies in rodents has been hampered by lack of PR3 expression on murine neutrophils, and by different Fc-receptor affinities for IgG across species. Therefore, we tested...

Journal: :Clinical and experimental immunology 2006
M Abdgawad T Hellmark L Gunnarsson K W A Westman M Segelmark

Several findings link proteinase 3 (PR3) to small vessel vasculitis. Besides being a major target of anti-neutrophil cytoplasm antibodies (ANCA), previous findings have shown increased circulating levels of PR3 in vasculitis patients, increased levels of neutrophil membrane-PR3 (mPR3) expression and a skewed distribution of the - 564 A/G polymorphism in the promotor region of the PR3 gene. In t...

Journal: :The Journal of clinical investigation 2015
Arnaud Millet Katherine R Martin Francis Bonnefoy Philippe Saas Julie Mocek Manal Alkan Benjamin Terrier Anja Kerstein Nicola Tamassia Senthil Kumaran Satyanarayanan Amiram Ariel Jean-Antoine Ribeil Loïc Guillevin Marco A Cassatella Antje Mueller Nathalie Thieblemont Peter Lamprecht Luc Mouthon Sylvain Perruche Véronique Witko-Sarsat

Granulomatosis with polyangiitis (GPA) is a systemic necrotizing vasculitis that is associated with granulomatous inflammation and the presence of anti-neutrophil cytoplasmic antibodies (ANCAs) directed against proteinase 3 (PR3). We previously determined that PR3 on the surface of apoptotic neutrophils interferes with induction of antiinflammatory mechanisms following phagocytosis of these cel...

Journal: :Annals of the rheumatic diseases 2007
Ymke M van der Geld Thomas Hellmark Daina Selga Peter Heeringa Minke G Huitema Pieter C Limburg Cees G M Kallenberg

AIM In this study, we employed chimeric human/mouse Proteinase 3 (PR3) proteins as tools to induce an autoantibody response to PR3 in rats and mice. METHOD Rats and mice were immunised with recombinant human PR3 (HPR3), recombinant murine PR3 (mPR3), single chimeric human/mouse PR3 (HHm, HmH, mHH, mmH, mHm, Hmm) or pools of chimeric proteins. Antibodies to mPR3 and HPR3 were measured by ELISA...

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