نتایج جستجو برای: ژن prnp

تعداد نتایج: 16699  

2014
Rita Guerreiro José Brás Aleksandra Wojtas Rosa Rademakers John Hardy Neill Graff-Radford

Here, we describe a nonsense haplotype in PRNP associated with clinical Alzheimer's disease. The patient presented an early-onset of cognitive decline with memory loss as the primary cognitive problem. Whole-exome sequencing revealed a nonsense mutation in PRNP (NM_000311, c.C478T; p.Q160*; rs80356711) associated with homozygosity for the V allele at position 129 of the protein, further highlig...

2010
Brenda M. Murdoch Michael L. Clawson Samuel Yue Urmila Basu Stephanie McKay Matthew Settles Rossana Capoferri William W. Laegreid John L. Williams Stephen S. Moore

BACKGROUND Classical bovine spongiform encephalopathy (BSE) is an acquired prion disease of cattle. The bovine prion gene (PRNP) contains regions of both high and low linkage disequilibrium (LD) that appear to be conserved across Bos taurus populations. The region of high LD, which spans the promoter and part of intron 2, contains polymorphic loci that have been associated with classical BSE st...

Journal: :PLoS ONE 2008
Michael L. Clawson Juergen A. Richt Thierry Baron Anne-Gaëlle Biacabe Stefanie Czub Michael P. Heaton Timothy P. L. Smith William W. Laegreid

BACKGROUND Atypical bovine spongiform encephalopathies (BSEs) are recently recognized prion diseases of cattle. Atypical BSEs are rare; approximately 30 cases have been identified worldwide. We tested prion gene (PRNP) haplotypes for an association with atypical BSE. METHODOLOGY/PRINCIPLE FINDINGS Haplotype tagging polymorphisms that characterize PRNP haplotypes from the promoter region throu...

Journal: :Neurology. Genetics 2016
Kagari Koshi Mano Takashi Matsukawa Jun Mitsui Hiroyuki Ishiura Shin-Ichi Tokushige Yuji Takahashi Naoko Saito Sato Fumiko Kusunoki Nakamoto Yaeko Ichikawa Yu Nagashima Yasuo Terao Jun Shimizu Masashi Hamada Yoshikazu Uesaka Genko Oyama Go Ogawa Jun Yoshimura Koichiro Doi Shinichi Morishita Shoji Tsuji Jun Goto

OBJECTIVE To delineate molecular and clinical characteristics of 3 families with PRNP P105L mutation, a variant of Gerstmann-Sträussler-Scheinker syndrome whose main motor symptoms were parkinsonism and/or involuntary movements. METHODS The causative mutation was first determined in the affected patients of family 1 using whole-exome sequencing, and then mutational analysis was extended to fa...

Journal: :American journal of physiology. Cell physiology 2006
Lorena Varela-Nallar Enrique M Toledo Luis F Larrondo Ana L B Cabral Vilma R Martins Nibaldo C Inestrosa

Prion diseases are caused by the conformational transition of the native alpha-helical cellular prion protein (PrPC) into a beta-sheet pathogenic isoform. However, the normal physiological function of PrPC remains elusive. We report herein that copper induces PrPC expression in primary hippocampal and cortical neurons. PrPC induced by copper has a normal glycosylation pattern, is proteinase K-s...

Journal: :Journal of Alzheimer's disease : JAD 2012
Enrico Premi Andrea Pilotto Antonella Alberici Alice Papetti Silvana Archetti Davide Seripa Antonio Daniele Carlo Masullo Valentina Garibotto Barbara Paghera Federico Caobelli Alessandro Padovani Barbara Borroni

Primary progressive aphasia (PPA) is a heterogeneous disorder characterized by progressive language impairment. Polymorphisms within forkhead box P2 gene (FOXP2) gene have been associated with speech and language impairment. Apolipoprotein E (APOE) genotype and PRNP 129 codon status have been demonstrated to increase the risk of PPA, but with contrasting results. In the present study, we have e...

2009
Gabriele Vaccari Cynthia H. Panagiotidis Cristina Acin Simone Peletto Francis Barillet Pierluigi Acutis Alex Bossers Jan Langeveld Lucien van Keulen Theodoros Sklaviadis Juan J. Badiola Olivier Andréoletti Martin H. Groschup Umberto Agrimi James Foster Wilfred Goldmann

Scrapie is a fatal, neurodegenerative disease of sheep and goats. It is also the earliest known member in the family of diseases classified as transmissible spongiform encephalopathies (TSE) or prion diseases, which includes Creutzfeldt-Jakob disease in humans, bovine spongiform encephalopathy (BSE), and chronic wasting disease in cervids. The recent revelation of naturally occurring BSE in a g...

2014
Hae-Young Shin Jeong-Ho Park Richard I. Carp Eun-Kyoung Choi Yong-Sun Kim

Normal cellular prion protein (PrP(C)) is highly expressed in the central nervous system. The Zürich I Prnp-deficient mouse strain did not show an abnormal phenotype in initial studies, however, in later studies, deficits in exploratory behavior and short- and long-term memory have been revealed. In the present study, numerous autophagic vacuoles were found in neurons from Zürich I Prnp-deficie...

2014
Keiji Uchiyama Hironori Miyata Masashi Yano Yoshitaka Yamaguchi Morikazu Imamura Naomi Muramatsu Nandita Rani Das Junji Chida Hideyuki Hara Suehiro Sakaguchi Noriyuki Nishida

Prion infection induces conformational conversion of the normal prion protein PrPC, into the pathogenic isoform PrPSc, in prion diseases. It has been shown that PrP-knockout (Prnp0/0) mice transgenically reconstituted with a mouse-hamster chimeric PrP lacking N-terminal residues 23-88, or Tg(MHM2Δ23-88)/Prnp 0/0 mice, neither developed the disease nor accumulated MHM2ScΔ23-88 in their brains af...

2010
Michael P Heaton Kreg A Leymaster Theodore S Kalbfleisch Brad A Freking Timothy PL Smith Michael L Clawson William W Laegreid

BACKGROUND Genetic predisposition to scrapie in sheep is associated with several variations in the peptide sequence of the prion protein gene (PRNP). DNA-based tests for scoring PRNP codons are essential tools for eradicating scrapie and for evaluating rare alleles for increased resistance to disease. In addition to those associated with scrapie, there are dozens more PRNP polymorphisms that ma...

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