نتایج جستجو برای: citrullinemia

تعداد نتایج: 234  

Journal: :International journal of molecular medicine 2011
Yuan-Zong Song Mei Deng Feng-Ping Chen Fang Wen Li Guo Shui-Liang Cao Jian Gong Hao Xu Guang-Yu Jiang Le Zhong Keiko Kobayashi Takeyori Saheki Zi-Neng Wang

Citrin is a liver-type aspartate/glutamate carrier (AGC) encoded by the gene SLC25A13. Two phenotypes for human citrin deficiency have been described, namely the adult-onset citrullinemia type II (CTLN2) and the neonatal intrahepatic cholestasis caused by citrin deficiency (NICCD). However, citrin deficiency currently remains a perplexing and poorly recognized disorder. In particular, descripti...

2013
Yuan-Zong Song Zhan-Hui Zhang Wei-Xia Lin Xin-Jing Zhao Mei Deng Yan-Li Ma Li Guo Feng-Ping Chen Xiao-Ling Long Xiang-Ling He Yoshihide Sunada Shun Soneda Akiko Nakatomi Sumito Dateki Lock-Hock Ngu Keiko Kobayashi Takeyori Saheki

BACKGROUND The human SLC25A13 gene encodes citrin, the liver-type mitochondrial aspartate/glutamate carrier isoform 2 (AGC2), and SLC25A13 mutations cause citrin deficiency (CD), a disease entity that encompasses different age-dependant clinical phenotypes such as Adult-onset Citrullinemia Type II (CTLN2) and Neonatal Intrahepatic Cholestasis caused by Citrin Deficiency (NICCD). The analyses of...

Journal: : 2021

A 49-year-old woman with intellectual disability and a food preference for fried chicken entered nursing home. After home diet, she developed episodic attacks of hyperammonemic encephalopathy. Her characteristic the negative results brain liver imaging studies suggested urea cycle disorder. high plasma citrulline level on amino acid analysis genetic test citrine gene confirmed deficiency (adult...

2016
Rachel Chan Jorge Sales Marques

Citrin deficiency (neonatal-onset type II citrullinemia), is an autosomal recessive metabolic disorder, caused by a mutation of the SLC25A13 gene. The defective transport between the mitochondria and the cytosol, leads to insufficient substrate for argininosuccinate synthetase (ASS) and secondary functional deficiency of ASS activity [1]. Infants with citrin deficiency, have transient intrahepa...

Journal: :International journal of molecular medicine 2007
Jung Min Ko Gu-Hwan Kim Ju-Hyun Kim Jae Young Kim Jin-Ho Choi Miharu Ushikai Takeyori Saheki Keiko Kobayashi Han-Wook Yoo

Citrin deficiency resulting from mutations of the SLC25A13 gene is associated with two major clinical phenotypes; neonatal intrahepatic cholestasis caused by citrin deficiency (NICCD) and adult-onset type 2 citrullinemia (CTLN2). In Korea, 6 cases of citrin deficiency were diagnosed based on biochemical and molecular findings. Four NICCD patients (2 boys and 2 girls) presented high citrulline l...

Journal: :Journal of chromatography. B, Analytical technologies in the biomedical and life sciences 2002
Yosuke Shigematsu Satoko Hirano Ikue Hata Yukie Tanaka Masakatsu Sudo Nobuo Sakura Tsuyoshi Tajima Seiji Yamaguchi

Electrospray tandem mass spectrometry was applied to detect a series of inherited metabolic disorders during a newborn-screening pilot study and a selective screening in Japan. In our mass screening of 102,200 newborns, five patients with propionic acidemia, two with methylmalonic acidemia, two with medium-chain acyl-CoA dehydrogenase deficiency, three with citrullinemia type II, and one with p...

Journal: :Molecular and cellular biology 2004
David S Sinasac Mitsuaki Moriyama M Abdul Jalil Laila Begum Meng Xian Li Mikio Iijima Masahisa Horiuchi Brian H Robinson Keiko Kobayashi Takeyori Saheki Lap-Chee Tsui

Adult-onset type II citrullinemia (CTLN2) is an autosomal recessive disease caused by mutations in SLC25A13, the gene encoding the mitochondrial aspartate/glutamate carrier citrin. The absence of citrin leads to a liver-specific, quantitative decrease of argininosuccinate synthetase (ASS), causing hyperammonemia and citrullinemia. To investigate the physiological role of citrin and the developm...

Journal: :Turkiye Klinikleri Journal of Neurology 2018

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