نتایج جستجو برای: hypermethylation

تعداد نتایج: 5378  

Journal: :Cancer research 2003
Ashok N Reddy Wei W Jiang Michael Kim Nicole Benoit Rodney Taylor John Clinger David Sidransky Joseph A Califano

A high frequency of death-associated protein kinase (DAPK) promoter hypermethylation has been noted in B-cell malignancies, head and neck cancers, and other solid tumors, and it has been used as a tumor marker in molecular detection strategies. Low levels of DAPK promoter hypermethylation, ranging from 0.003 to 1.181%, were detected in peripheral blood cells from 75 of 143 (52%) normal subjects...

2012
Ho-Jin Kang Eun-Jeong Kim Byoung-Gwon Kim Chang-Hun You Sang-Yong Lee Dong-Il Kim Young-Seoub Hong

OBJECTIVES The purpose of this paper was to elucidate the potential methylation levels of adjacent normal and cancer tissues by comparing them with normal colorectal tissues, and to describe the correlations between the methylation and clinical parameters in Korean colorectal cancer (CRC) patients. METHODS Hypermethylation profiles of nine genes (RASSF1, APC, p16(INK4a), Twist1, E-cadherin, T...

2017
Huiqing Yu Liejun Yang Yunfeng Fu Meng Gao Ling Tian

It is well known that the loss of function of the p16INK4A gene is mainly caused by the hypermethylation of the p16 gene; however, whether or not the inactivation is associated with the clinical significance of multiple myeloma (MM) remains elusive. A meta-analysis was conducted to quantitatively determine the role of the p16 hypermethylation in the clinical significance of MM. We demonstrated ...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2007
Gregory R Bean Andrew D Bryson Patrick G Pilie Vanessa Goldenberg Joseph C Baker Catherine Ibarra Danielle M U Brander Carolyn Paisie Natalie R Case Mona Gauthier Paul A Reynolds Eric Dietze Julie Ostrander Victoria Scott Lee G Wilke Lisa Yee Bruce F Kimler Carol J Fabian Carola M Zalles Gloria Broadwater Thea D Tlsty Victoria L Seewaldt

PURPOSE p16(INK4a) has been appreciated as a key regulator of cell cycle progression and senescence. Cultured human mammary epithelial cells that lack p16(INK4a) activity have been shown to exhibit premalignant phenotypes, such as telomeric dysfunction, centrosomal dysfunction, a sustained stress response, and, most recently, a dysregulation of chromatin remodeling and DNA methylation. These da...

Journal: :International journal of cancer 2006
David Shibata Yuriko Mori Kun Cai Li Zhang Jing Yin Abul Elahi Richard Hamelin Yick F Wong Wing K Lo Tony K H Chung Fumiaki Sato Martin S Karpeh Stephen J Meltzer

The recently described gene, RAB32, is a ras proto-oncogene family member that encodes an A-kinase-anchoring protein. RAB32 has been found to be frequently hypermethylated in microsatellite instability-high (MSI-H) colon cancers. We sought to determine the prevalence of RAB32 hypermethylation in gastric and endometrial adenocarcinomas, the 2 other major tumor types in which MSI-H is common. Mor...

Journal: :iranian biomedical journal 0
مژگان راستی mozhgan rasti پرستو توسلی parastoo tavasoli احمد منابتی ahmad monabati مونا انتظام mona entezam

background: ras-associated domain family 1 (rassf1a) and hypermethylated in cancer (hic1) genes are methylated more frequently in breast cancer. genetic factors that alter the dna methylation levels in normal and tumor tissues could therefore influence the susceptibility to this tumor phenotype. objective: we determined the frequency of aberrant methylation of hic1 and rassf1a gene promoters an...

2015
Zhidong Wang Bin Wang Huanchen Guo Guoyu Shi Xiuqin Hong

BACKGROUND Previous studies demonstrate that T-cadherin is a candidate tumor suppressor in several types of human tumors, including non-small cell lung cancer (NSCLC). Lack of protein expression of T-cadherin by hypermethylation has been found to play an important role in lung alveolar differentiation regulation and epithelial tumorigenesis. However, the correlation between T-cadherin hypermeth...

Journal: :Oncology reports 2007
Megumi Yanokura Kouji Banno Makiko Kawaguchi Nobumaru Hirao Akira Hirasawa Nobuyuki Susumu Katsumi Tsukazaki Daisuke Aoki

The relationship of aberrant DNA hypermethylation of cell cycle checkpoint genes with the sensitivity of cancer cells to anticancer drugs is a question of current interest. In this study, we investigated the relationship between aberrant hypermethylation of the CHFR (checkpoint with forkhead-associated and ring finger) mitotic checkpoint gene and sensitivity to taxanes in endometrial cancer. Me...

Journal: :Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology 2008
JianMing Wang Annie J Sasco ChaoWei Fu HengChuan Xue GuoPing Guo ZhaoLai Hua Qing Zhou QingWu Jiang Biao Xu

To explore the role of aberrant hypermethylation of cancer-related genes, such as P16, MGMT, and hMLH1, in the esophageal squamous cell carcinoma (ESCC) as well as its relation to dietary folate intake and MTHFR C677T polymorphism, we conducted a molecular epidemiologic study in China. One hundred and twenty-five histologically confirmed ESCC patients having undergone surgery in the Yangzhong P...

Journal: :Human molecular genetics 2001
D J Smiraglia L J Rush M C Frühwald Z Dai W A Held J F Costello J C Lang C Eng B Li F A Wright M A Caligiuri C Plass

Cancer cell lines are widely used in many types of cancer research, including studies aimed at understanding DNA hypermethylation of gene promoters in cancer. Hypermethylation of promoters is capable of repressing the expression of tumor suppressor genes and may play a role in the development and/or progression of cancer. Although both primary malignancies and cancer cell lines exhibit this epi...

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