نتایج جستجو برای: tp53 mutation and expression

تعداد نتایج: 16973495  

2017
Alexandra J van den Broek Annegien Broeks Hugo M Horlings Sander VM Canisius Linde M Braaf Anita Langerød Laura J Van ’t Veer Marjanka K Schmidt

assoCIatIon of the GermlIne MDM2 snp309 and TP53 r72p varIants WIth breast CanCer survival in sPeciFic tumor subgrouPs. absTRacT The tumor suppressor gene TP53 and its regulator MDM2 are both important players in the DNA-damage repair 'TP53 response pathway'. Common germline polymorphisms in these genes may affect outcome in patients with tumors characterized by additional somatic changes in th...

Journal: :Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology 2005
John K Wiencke Kenneth Aldape Alex McMillan Joe Wiemels Michelle Moghadassi Rei Miike Karl T Kelsey Joe Patoka Jeff Long Margaret Wrensch

BACKGROUND Risk factors for adult glioma in the San Francisco Bay Area include well-known demographic features such as age and race/ethnicity, and our previous studies indicated that these characteristics are associated with the TP53 mutation status of patients' tumors. We enlarged our study to assess the relationships of risk factors with TP53 as well as epidermal growth factor receptor (EGFR)...

Journal: :Blood 2013
Annika Dufour Giuseppe Palermo Evelyn Zellmeier Gudrun Mellert Guillemette Duchateau-Nguyen Stephanie Schneider Tobias Benthaus Purvi M Kakadia Karsten Spiekermann Wolfgang Hiddemann Jan Braess Sim Truong Nancy Patten Lin Wu Sabine Lohmann David Dornan Debraj GuhaThakurta Ru-Fang Yeh Galina Salogub Philippe Solal-Celigny Anna Dmoszynska Tadeusz Robak Marco Montillo John Catalano Christian H Geisler Martin Weisser Stefan K Bohlander

In chronic lymphocytic leukemia (CLL) patients, disruptions of the TP53 tumor suppressor pathway by 17p13 deletion (del17p), somatic TP53 mutations, or downregulation of microRNA-34a have been associated with a poor prognosis. So far, the impact of the various TP53 defects has not been evaluated in a large cohort of previously treated and relapsed CLL patients. Here, we present the results of T...

Journal: :Clinical chemistry 2009
Sandra Regina Jean-Baptiste Valentin Sébastien Lachot Etienne Lemarié Jérôme Rollin Yves Gruel

BACKGROUND Tissue factor (TF), the main initiator of blood coagulation, is also a signaling protein that regulates cancer progression. TF synthesis was recently shown to be affected by tumor suppressor genes (TSGs) in tumor cell lines. We therefore studied TF gene (F3) expression and the status of genes coding for tumor protein p53 (TP53), phosphatase and tensin homolog (PTEN), and serine/threo...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2011
Daisuke Sano Tong-Xin Xie Thomas J Ow Mei Zhao Curtis R Pickering Ge Zhou Vlad C Sandulache David A Wheeler Richard A Gibbs Carlos Caulin Jeffrey N Myers

PURPOSE To characterize tumor growth and metastatic potential in head and neck squamous cell carcinoma (HNSCC) cell lines in an orthotopic murine model of oral tongue cancer and to correlate TP53 mutation status with these findings. EXPERIMENTAL DESIGN Cells from each of 48 HNSCC cell lines were orthotopically injected into the oral tongues of nude mice. Tumor volume, cervical lymph node meta...

Journal: :Radiotherapy and oncology : journal of the European Society for Therapeutic Radiology and Oncology 2006
Aslaug Helland Hilde Johnsen Caroline Frøyland Hege B K Landmark Anna B Saetersdal Marit Muri Holmen Turid Gjertsen Jahn M Nesland William Ottestad Stefanie S Jeffrey Lars O Ottestad Olag K Rodningen Gavin Sherlock Anne-Lise Børresen-Dale

BACKGROUND AND PURPOSE Breast cancer is diagnosed worldwide in approximately one million women annually and radiation therapy is an integral part of treatment. The purpose of this study was to investigate the molecular basis underlying response to radiotherapy in breast cancer tissue. MATERIAL AND METHODS Tumour biopsies were sampled before radiation and after 10 treatments (of 2 Gray (Gy) ea...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2005
Florian Haller Bastian Gunawan Anja von Heydebreck Stefanie Schwager Hans-Jürgen Schulten Judith Wolf-Salgó Claus Langer Giuliano Ramadori Holger Sültmann László Füzesi

PURPOSE The aim of the current study was to examine the prognostic relevance of the CDKN2A tumor suppressor pathway in gastrointestinal stromal tumors (GIST). EXPERIMENTAL DESIGN We determined the mRNA expression of p1(INK4A), p14(ARF), CDK4, RB1, MDM2, TP53, and E2F1 by quantitative reverse transcription-PCR in 38 cases of GISTs and correlated the findings with clinicopathologic factors, inc...

Journal: :Carcinogenesis 2005
Leah E Mechanic Aizen J Marrogi Judith A Welsh Elise D Bowman Mohammed A Khan Lindsey Enewold Yun-Ling Zheng Stephen Chanock Peter G Shields Curtis C Harris

The pattern of somatic mutations in TP53 is distinct for particular cancers and carcinogenic exposures, providing clues to disease etiology, e.g. G:C-->T:A mutations in TP53 are more frequently observed in smoking-associated lung cancers. In order to investigate possible causes and mechanisms of lung cancer susceptibility differences, the TP53 gene was sequenced in a case-only study of lung can...

Journal: :Pathology international 2015
Takuro Nakazawa Sumihito Nobusawa Hayato Ikota Hiroyuki Kuwano Izumi Takeyoshi Hideaki Yokoo

The pathogenesis of sarcomatous component in spindle cell carcinoma (SpCC) of the esophagus is unclear. To investigate the involvement of epithelial-mesenchymal transition (EMT) in sarcomatous differentiation, we performed immunohistochemistry for Slug, Twist, ZEB1, and ZEB2, transcription factors associated with EMT and E-cadherin, in 14 cases of SpCC of the esophagus. In order to verify the n...

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