نتایج جستجو برای: bad gene

تعداد نتایج: 1170207  

Journal: :research in pharmaceutical sciences 0
parham reisi 1department of physiology, school of medicine, isfahan university of medical sciences, isfahan, i.r. iran. nastaran eidelkhani 1department of physiology, school of medicine, isfahan university of medical sciences, isfahan, i.r. iran. laleh rafiee 2applied physiology research center, isfahan university of medical sciences, isfahan, i.r. iran. mohammad kazemi 3department of genetics and molecular biology, school of medicine, isfahan university of medical sciences, isfahan, i.r. iran. maryam radahmadi 1department of physiology, school of medicine, isfahan university of medical sciences, isfahan, i.r. iran. hojjatallah alaei 1department of physiology, school of medicine, isfahan university of medical sciences, isfahan, i.r. iran.

stress is one of the effective factors in the development of depressive disorders that performs some parts of its effects by affecting hippocampus. since doxepin has been shown to have neuroprotective effects, in this study, we focused on the effects of doxepin on the expression of involved genes in neuronal survival and plasticity in the rat hippocampus following chronic stress. male wistar ra...

Journal: :Journal of Oncology 2021

Esophageal cancer (EC) is the eighth most prevalent and sixth leading cause of cancer-related mortality worldwide. As an antiapoptotic a proapoptotic protein, respectively, survivin Bad play important role in carcinogenesis human cancers including EC. However, regulatory relationships between them remain unclear. We sought to investigate effects knockdown overexpression on expression gene, cell...

Journal: :British Journal of General Practice 2016

Journal: :Journal of virology 2016
Hyoji Kim Hoyun Choi Suk Kyeong Lee

UNLABELLED Epstein-Barr virus (EBV) is a human gammaherpesvirus associated with a variety of tumor types. EBV can establish latency or undergo lytic replication in host cells. In general, EBV remains latent in tumors and expresses a limited repertoire of latent proteins to avoid host immune surveillance. When the lytic cycle is triggered by some as-yet-unknown form of stimulation, lytic gene ex...

2001
C. FERNANDES

In this paper we describe a method for generating high school timetables using an Evolutionary Algorithm (E.A.) with a new set of genetic operators inspired on the Infected Genes Evolutionary Algorithm [14]. We show that widening region of infection considered in [7] leads to a very significant increase in the performance of the E.A.. Comparative tests between the E.A. with and without the impr...

1999
Carlos M. Fernandes João Paulo Caldeira Fernando Melicio Agostinho C. Rosa

In this paper we describe a method for generating high school timetables using an Evolutionary Algorithm (E.A.) with a new set of genetic operators inspired on the Infected Genes Evolutionary Algorithm (Tavares, 99). We show that widening region of infection considered in (Fernandes, 99) leads to a very significant increase in the performance of the E.A.. Comparative tests between the E.A. with...

2015
XIAOMANG B STICKLES DOUGLAS C MARCHION ELONA BICAKU ENTIDHAR AL SAWAH FOROUGH ABBASI YIN XIONG NADIM BOU ZGHEIB BERNADETTE M BOAC BRIAN C ORR PATRICIA L JUDSON AMY BERRY ARDESHIR HAKAM ROBERT M WENHAM SACHIN M APTE ANDERS E BERGLUND JOHNATHAN M LANCASTER

The malignant transformation of normal cells is caused in part by aberrant gene expression disrupting the regulation of cell proliferation, apoptosis, senescence and DNA repair. Evidence suggests that the Bcl-2 antagonist of cell death (BAD)-mediated apoptotic pathway influences cancer chemoresistance. In the present study, we explored the role of the BAD-mediated apoptotic pathway in the devel...

Journal: :The Journal of Cell Biology 2003
William A. Wells

utrition and cell death are integrated in a single mitochondrial complex, according to Nika Danial, Stanley Korsmeyer (Harvard Medical School, Boston, MA), and colleagues. The complex was discovered after gel filtration of the pro-apoptotic BAD protein, and contains BAD, a phosphatase, kinase, and kinase-anchoring protein directed at BAD, and glucokinase. In cells lacking BAD, the complex falls...

2011
Douglas C. Marchion Hope M. Cottrill Yin Xiong Ning Chen Elona Bicaku William J. Fulp Nisha Bansal Hye Sook Chon Xiaomang B. Stickles Siddharth G. Kamath Ardeshir Hakam Lihua Li Dan Su Carolina Moreno Patricia L. Judson Andrew Berchuck Robert M. Wenham Sachin M. Apte Jesus Gonzalez-Bosquet Gregory C. Bloom Steven A. Eschrich Said Sebti Dung-Tsa Chen Johnathan M. Lancaster

Purpose: Despite initial sensitivity to chemotherapy, ovarian cancers (OVCA) often develop drug-resistance, which limits patient survival. Using specimens and/or genomic data from 289 patients and a panel of cancer cell lines, we explored genome-wide expression changes that underlie the evolution of OVCA chemo-resistance and characterized the BCL2 antagonist of cell death (BAD) apoptosis pathwa...

2011
Douglas C. Marchion Hope M. Cottrill Yin Xiong Ning Chen Elona Bicaku William J. Fulp Nisha Bansal Hye Sook Chon Xiaomang B. Stickles Siddharth G. Kamath Ardeshir Hakam Lihua Li Dan Su Carolina Moreno Patricia L. Judson Andrew Berchuck Robert M. Wenham Sachin M. Apte Jesus Gonzalez-Bosquet Gregory C. Bloom Steven A. Eschrich Said Sebti Dung-Tsa Chen Johnathan M. Lancaster

Purpose: Despite initial sensitivity to chemotherapy, ovarian cancers (OVCA) often develop drug-resistance, which limits patient survival. Using specimens and/or genomic data from 289 patients and a panel of cancer cell lines, we explored genome-wide expression changes that underlie the evolution of OVCA chemo-resistance and characterized the BCL2 antagonist of cell death (BAD) apoptosis pathwa...

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