نتایج جستجو برای: conjugated hyperbilirubinemia

تعداد نتایج: 30488  

2017
Ryoichi Fujiwara Ryo Mitsugi Asuka Uemura Tomoo Itoh Robert H. Tukey

Neurotoxic bilirubin is solely conjugated by UDP-glucuronosyltransferase (UGT) 1A1. Due to an inadequate function of UGT1A1, human neonates develop mild to severe physiological hyperbilirubinemia. Accumulation of bilirubin in the brain leads to the onset of irreversible brain damage called kernicterus. Breastfeeding is one of the most significant factors that increase the risk of developing ker...

Journal: :Drug metabolism and pharmacokinetics 2009
Daisuke Kanda Hitoshi Takagi Yasutsugu Kawahara Yutaka Yata Tomofumi Takakusagi Takeshi Hatanaka Teruo Yoshinaga Keigo Iesaki Kenji Kashiwabara Tsugio Higuchi Masatomo Mori Takeshi Hirota Shun Higuchi Ichiro Ieiri

The Dubin-Johnson syndrome (DJS) is an inherited liver disorder characterized by conjugated hyperbilirubinemia and caused by ABCC2 gene mutations resulting in deficiency of multidrug resistance associated-protein 2 (MRP2) function. A 76-year-old woman with serious jaundice was referred to our hospital. She was clinically diagnosed with DJS with hepatic congestion, due to constrictive pericardit...

2013
Rajendra Prasad Ritu Rathi Praveen Kumar

Hyperbilirubinemia is the most common problem encountered in terms newborns which is mainly caused by abnormal liver function, hemolysis or genetic defect. Uncongugated bilirubin is produced mainly by the turnover of erythrocytes, after that it is transported by organic anion transporter polypeptide (OATP) to the liver, where uncongugated bilirubin is conjugated by uridine diphosphoglucuronate ...

Journal: :Canadian Journal of Gastroenterology and Hepatology 2021

2014
Dietrich Keppler

Increased concentrations of bilirubin glucuronides in blood plasma indicate hepatocellular dysfunction. Elucidation of the transport processes of bilirubin conjugates across the basolateral (sinusoidal) and the canalicular plasma membrane domains of hepatocytes has decisively contributed to our current understanding of the molecular basis of conjugated hyperbilirubinemia in human liver diseases...

Journal: :Drug metabolism and disposition: the biological fate of chemicals 2014
Dietrich Keppler

Increased concentrations of bilirubin glucuronides in blood plasma indicate hepatocellular dysfunction. Elucidation of the transport processes of bilirubin conjugates across the basolateral (sinusoidal) and the canalicular plasma membrane domains of hepatocytes has decisively contributed to our current understanding of the molecular basis of conjugated hyperbilirubinemia in human liver diseases...

2017
Fang Kuan Chiou Christina Ong Kong Boo Phua Fares Chedid Ajmal Kader

AIM To describe the etiology and characteristics of early-onset conjugated hyperbilirubinemia (ECHB) presenting within 14 d of life in term neonates. METHODS Retrospective review was performed of term infants up to 28-d-old who presented with conjugated hyperbilirubinemia (CHB) at a tertiary center over a 5-year period from January 2010 to December 2014. CHB is defined as conjugated bilirubin...

Journal: :Annals of clinical and laboratory science 1984
E Kahn F Daum

Syndromatic paucity of interlobular bile ducts is a common cause of conjugated hyperbilirubinemia in children. The clinical presentation is not always obvious. Therefore, the liver biopsy may be a useful diagnostic tool in the definition of this entity. The hepatic and biliary morphology of five children with arteriohepatic dysplasia (Allagille' syndrome) is described. Prior to diagnosis, four ...

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