نتایج جستجو برای: gluten ataxia
تعداد نتایج: 24312 فیلتر نتایج به سال:
Neurologic complications of celiac disease (CD) include ataxia and peripheral neuropathy, which can be the presenting symptoms and signs. Early diagnosis and intervention could prevent development of further neurologic and systemic complications. Questions remain regarding the prevalence of the neurologic complications, the pathophysiological mechanisms, and the effectiveness of therapy or resp...
A decade ago celiac disease was considered extremely rare outside Europe and, therefore, was almost completely ignored by health care professionals. In only 10 years, key milestones have moved celiac disease from obscurity into the popular spotlight worldwide. Now we are observing another interesting phenomenon that is generating great confusion among health care professionals. The number of in...
IMPORTANCE Celiac disease is an autoimmune disorder triggered by gluten in genetically predisposed individuals. Gluten sensitivity can cause neurologic manifestations, such as ataxia or neuropathy, with or without gastrointestinal symptoms. Many patients with gluten ataxia produce antibodies toward the newly identified neuronal transglutaminase 6 (TG6). Two case reports described patients initi...
We have previously shown that 67% of patients with newly diagnosed coeliac disease (CD) presenting to gastroenterologists evidence neurological dysfunction. This manifested headache and loss co-ordination. Furthermore 60% these had abnormal brain imaging. In this follow-up study, we re-examined re-scanned 30 from the original cohort 100, seven years later. There was significant reduction in pre...
OBJECTIVE Celiac disease (CD), or gluten sensitivity, is considered to be a state of heightened immunologic responsiveness to ingested gluten proteins in genetically predisposed individuals. The gastrointestinal manifestation suggests a severe enteropathy of the small intestine with malabsorption, steatorrhea, and weight loss because of a deranged mucosal immune response. Neurologic complicatio...
Since the first description of immune-mediated cerebellar ataxias (IMCAs) by Charcot in 1868, several milestones have been reached our understanding this group neurological disorders. IMCAs diverse etiologies, such as gluten ataxia, postinfectious cerebellitis, paraneoplastic degeneration, opsoclonus myoclonus syndrome, anti-GAD and primary autoimmune ataxia. The cerebellum, a vulnerable target...
Gluten can cause extraintestinal manifestations with or without gastrointestinal symptoms and elevated antitissue transglutaminase 2 (tTG2) autoantibodies. Organ-specific gluten reaction involves immune response toward other transglutaminase (TG) isoforms including tTG3 (expressed in the skin, leading to dermatitis herpetiformis) and tTG6 (expressed in the brain, causing gluten ataxia). This an...
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