نتایج جستجو برای: hdac inhibitor

تعداد نتایج: 213269  

2005
Søren Skov Marianne Terndrup Pedersen Lars Andresen Per Thor Straten Anders Woetmann Niels Ødum

We show that histone deacetylase (HDAC) inhibitors lead to functional expression of MHC class I–related chain A and B (MICA/B) on cancer cells, making them potent targets for natural killer (NK) cell–mediated killing through a NK group 2, member D (NKG2D) restricted mechanism. Blocking either apoptosis or oxidative stress caused by HDAC inhibitor treatment did not affect MICA/B expression, sugg...

Journal: :EMBO Reports 2009
Angela Nebbioso Fabio Manzo Marco Miceli Mariarosaria Conte Lucrezia Manente Alfonso Baldi Antonio De Luca Dante Rotili Sergio Valente Antonello Mai Alessandro Usiello Hinrich Gronemeyer Lucia Altucci

Histone deacetylase (HDAC) inhibitors are promising new epi-drugs, but the presence of both class I and class II enzymes in HDAC complexes precludes a detailed elucidation of the individual HDAC functions. By using the class II-specific HDAC inhibitor MC1568, we separated class I- and class II-dependent effects and defined the roles of class II enzymes in muscle differentiation in cultured cell...

Journal: :Molecular cancer therapeutics 2010
Jinkoo Kim Jean Guan Insoon Chang Xiaohong Chen Demin Han Cun-Yu Wang

Proteasome inhibitor PS-341 (also known as bortezomib) and histone deacetylase (HDAC) inhibitors have emerged as novel therapeutic agents for a variety of malignancies. In this study, we examined whether PS-341 and the HDAC inhibitor trichostatin A (TSA) induced apoptosis in head and neck squamous cell carcinoma (HNSCC), a common and lethal malignancy. We found that, although TSA treatment alon...

2012
Tom Latham Logan Mackay Duncan Sproul Muhammed Karim Jayne Culley David J Harrison Larry Hayward Pat Langridge-Smith Nick Gilbert Bernard H Ramsahoye

Chemical inhibitors of histone deacetylase (HDAC) activity are used as experimental tools to induce histone hyperacetylation and deregulate gene transcription, but it is not known whether the inhibition of HDACs plays any part in the normal physiological regulation of transcription. Using both in vitro and in vivo assays, we show that lactate, which accumulates when glycolysis exceeds the cell'...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2004
Xin-Yan Pei Yun Dai Steven Grant

PURPOSE The purpose of this study was to examine interactions between the proteasome inhibitor bortezomib (Velcade) and the histone deacetylase (HDAC) inhibitors sodium butyrate and suberoylanilide hydroxamic acid in human multiple myeloma (MM) cells that are sensitive and resistant to conventional agents. EXPERIMENTAL DESIGN MM cells were exposed to bortezomib for 6 h before the addition of ...

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