نتایج جستجو برای: il 31

تعداد نتایج: 283590  

2012
Albert Duschl Angelika Stoecklinger Thomas Hawranek Elisabeth Maier Stefan Hainzl Maria Schmittner Jutta Horejs-Hoeck Harald Schwarz Sebastian Lamprecht Gernot Posselt

Journal: :Journal of Investigative Dermatology 2023

In this study, we aimed at characterizing the dominant cell source of IL-31 and its detailed phenotype, which is still a matter debate. We used flow cytometry as well bulk single RNA sequencing datasets to identify main IL-31, phenotyped circulating IL-31- producing T cells. CD4+CD45RO+ memory cells represent IL31-producing leukocyte subset within circulation in healthy donors. Patients with at...

Journal: :European annals of allergy and clinical immunology 2015
F Guarneri P L Minciullo C Mannucci F Calapai S Saitta S P Cannavò S Gangemi

Enhanced IL-31 expression in skin biopsies is present in allergic contact dermatitis (ACD). IL-33 expression is induced in keratinocytes and in skin of ACD patients. This overexpression is present in both allergic and irritant conditions. The aim of this work was to test the systemic involvement of IL-31 and IL-33 in ACD. IL-31 levels were significantly higher in patients than in controls. IL-3...

Journal: :Acta dermato-venereologica 2010
Nikoletta Nagy Akio Tanaka Tanasit Techanukul John A McGrath

Epidermolysis bullosa pruriginosa (EBP; OMIM #604129) is an unusual variant of autosomal dominant (or occasionally recessive) dystrophic epidermolysis bullosa (DEB) in which intense itching and scratching impacts upon the phenotype (1, 2). Although trauma-induced blistering often occurs, and toenail dystrophy is almost universal, the skin lesions can often resemble nodular prurigo, lichen simpl...

2012
Chun-Kwok Wong Karen Ming-Lam Leung Huai-Na Qiu Joyce Yin-Sau Chow Angela On Kei Choi Christopher Wai-Kei Lam

BACKGROUND IL-31 is a pruritogenic cytokine, and IL-33 is an alarmin for damaging inflammation. They together relate to the pathogenesis of atopic dermatitis (AD). Eosinophil infiltration into the inner dermal compartment is a predominant pathological feature of AD. We herein investigated the in vitro inflammatory effects of IL-31 and IL-33 on the activation of human eosinophils and dermal fibr...

2017
Elisa Ferretti Stefan Hohaus Arianna Di Napoli Beatrice Belmonte Annarosa Cuccaro Elisa Cupelli Eugenio Galli Vittoria Rufini Gino Tripodi Giulio Fraternali-Orcioni Vito Pistoia Anna Corcione

Hodgkin Lymphoma (HL) is a tumor of B-cell origin characterized by Hodgkin and Reed-Stenberg (H/RS) cells embedded in an inflammatory tissue where numerous cytokines/chemokines contribute to shape the microenvironment, leading to the typical clinical symptoms. We investigated: i) the expression of Interleukin-IL-31 (IL-31) and Thymic Stromal Lymphopoietin (TSLP), two Th2-related cytokines with ...

2017
Deependra Kumar Yadav Anil Kumar Tripathi Divya Gupta Saurabh Shukla Aloukick Kumar Singh Ashutosh Kumar Jyotsna Agarwal K. N. Prasad

Background Immune thrombocytopenia (ITP) is an immune-mediated disease caused by autoantibodies against platelets membrane glycoproteins GPIIb/IIIa and GPIb/IX. The etiology of ITP remains unclear. This study evaluated the association of polymorphisms in interleukin (IL)-1B-31, IL-1B-511, and IL-1Ra with ITP. Methods Genotyping of IL-1B-31, IL-1B-511, and IL-1Ra was performed in 118 ITP patie...

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