Pseudoaneurysm of the left ventricle is rare and may occur as a result of transmural myocardial infarction. The course of rupture after acute myocardial infarction varies from a catastrophic event, with an acute tear leading to immediate death (acute rupture), or slow and incomplete tear leading to a late rupture (subacute rupture). Incomplete rupture may occur when the thrombus and haematoma t...

BACKGROUND Takostsubo (stress) cardiomyopathy (TC) is a clinical syndrome featuring transient left ventricular dysfunction and wall-motion abnormalities, usually following emotional or physical stress. The diagnosis of TC depends on fulfillment of multiple clinical criteria. Although the pathogenesis has not been firmly established, myocardial cathecholamine toxicity is thought to represent a p...

coronary artery perforation is a rare, but potentially serious, complication of percutaneous coronary intervention and is associated with a high incidence of death, q-wave myocardial infarction, and emergency coronary bypass surgery. management is different but requires prompt recognition and treatment. we describe a case of coronary perforation only after guidewire removal and also review the ...

The clinical features of cardiac rupture with tamponade in acute myocardial in-farction have been studied in an attempt to predict this complication and find clues to prophylactic and therapeutic measures. Eleven cases with rupture of the free left ventricular wall in a consecutive series of 529 coronary care unit (CCU) patients with acute myocardial infarction have been analyzed. These 11 pati...

The 10-20% incidence of cardiac rupture in acute fatal myocardial infarction has not changed in the past century, and little is known about its pathophysiology. To determine whether expansion acute dilatation and thinning of the area of infarction not explained by resorption of necrotic tissue may be a variable predictive for rupture, we studied 110 consecutive autopsied patients who died of ac...

The 10-20% incidence of cardiac rupture in acute fatal myocardial infarction has not changed in the past century, and little is known about its pathophysiology. To determine whether expansion acute dilatation and thinning of the area of infarction not explained by resorption of necrotic tissue may be a variable predictive for rupture, we studied 110 consecutive autopsied patients who died of ac...