نتایج جستجو برای: pkcγ

تعداد نتایج: 103  

Journal: :Cell reports 2015
Hugues Petitjean Sophie Anne Pawlowski Steven Li Fraine Behrang Sharif Doulia Hamad Tarheen Fatima Jim Berg Claire M Brown Lily-Yeh Jan Alfredo Ribeiro-da-Silva Joao M Braz Allan I Basbaum Reza Sharif-Naeini

Neuropathic pain is a chronic debilitating disease that results from nerve damage, persists long after the injury has subsided, and is characterized by spontaneous pain and mechanical hypersensitivity. Although loss of inhibitory tone in the dorsal horn of the spinal cord is a major contributor to neuropathic pain, the molecular and cellular mechanisms underlying this disinhibition are unclear....

2015
Pau Creixell Erwin M. Schoof Craig D. Simpson James Longden Chad J. Miller Hua Jane Lou Lara Perryman Thomas R. Cox Nevena Zivanovic Antonio Palmeri Agata Wesolowska-Andersen Manuela Helmer-Citterich Jesper Ferkinghoff-Borg Hiroaki Itamochi Bernd Bodenmiller Janine T. Erler Benjamin E. Turk Rune Linding

Cancer cells acquire pathological phenotypes through accumulation of mutations that perturb signaling networks. However, global analysis of these events is currently limited. Here, we identify six types of network-attacking mutations (NAMs), including changes in kinase and SH2 modulation, network rewiring, and the genesis and extinction of phosphorylation sites. We developed a computational pla...

Journal: :Biological & pharmaceutical bulletin 2011
Hiroshi Ueda

The injury-induced intense stimulation of spinal cord neurons causes lysophosphatidic acid (LPA) biosynthesis. LPA(1) receptor activation causes demyelination and sprouting of dorsal root fibers, leading to an induction of synaptic reorganization underlying allodynia, in which innocuous (tactile) stimuli cause intense pain. The LPA(1) signal also initiates the up-regulation of Ca(v)α2δ1 in dors...

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