نتایج جستجو برای: sporadic amyotrophic lateral sclerosis sals

تعداد نتایج: 198646  

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2007
Arie Gruzman William L Wood Evgenia Alpert M Dharma Prasad Robert G Miller Jeffery D Rothstein Robert Bowser Ronald Hamilton Troy D Wood Don W Cleveland Vishwanath R Lingappa Jian Liu

Amyotrophic lateral sclerosis (ALS) is a devastating motor neuron degenerative disease whose etiology and pathogenesis remain poorly understood. Most cases of ALS ( approximately 90%) are sporadic (SALS), occurring in the absence of genetic associations. Approximately 20% of familial ALS (FALS) cases are due to known mutations in the copper, zinc superoxide dismutase (SOD1) gene. Molecular evid...

2013
Xianghua He Lifang Zhang Xiaoli Yao Jing Hu Lihua Yu Hua Jia Ran An Zhuolin Liu Yanming Xu

Parkinson's disease (PD) and amyotrophic lateral sclerosis (ALS) share several clinical and neuropathologic features, and studies suggest that several gene mutations and polymorphisms are involved in both conditions. Matrix metalloproteinase-9 (MMP-9) is implicated in the pathogenesis of PD and ALS, and the C(-1562)T polymorphism in the MMP-9 gene leads to higher promoter activity. We therefore...

2014
Svitlana Garbuzova-Davis Paul R. Sanberg

Amyotrophic lateral sclerosis (ALS) is a severe neurodegenerative disease with a complicated and poorly understood pathogenesis. Recently, alterations in the blood-Central Nervous System barrier (B-CNS-B) have been recognized as a key factor possibly aggravating motor neuron damage. The majority of findings on ALS microvascular pathology have been determined in mutant superoxide dismutase (SOD1...

Journal: :JAMA neurology 2015
Nimeshan Geevasinga Parvathi Menon James Howells Garth A Nicholson Matthew C Kiernan Steve Vucic

IMPORTANCE Abnormalities of axonal excitability characterized by upregulation of persistent sodium (Na+) conductances and reduced potassium (K+) currents have been reported in sporadic amyotrophic lateral sclerosis (SALS) phenotypes and linked to the development of clinical features such as fasciculations and neurodegeneration. OBJECTIVE To investigate whether abnormalities of axonal ion chan...

Journal: :Human molecular genetics 2009
Mario Sabatelli Fabrizio Eusebi Ammar Al-Chalabi Amelia Conte Francesca Madia Marco Luigetti Irene Mancuso Cristina Limatola Flavia Trettel Fabrizia Sobrero Silvia Di Angelantonio Francesca Grassi Amalia Di Castro Claudia Moriconi Sergio Fucile Serena Lattante Giuseppe Marangi Marina Murdolo Daniela Orteschi Alessandra Del Grande Pietro Tonali Giovanni Neri Marcella Zollino

Sporadic amyotrophic lateral sclerosis (SALS) is a motor neuron degenerative disease of unknown etiology. Current thinking on SALS is that multiple genetic and environmental factors contribute to disease liability. Since neuronal acetylcholine receptors (nAChRs) are part of the glutamatergic pathway, we searched for sequence variants in CHRNA3, CHRNA4 and CHRNB4 genes, encoding neuronal nicotin...

Journal: :Neurology 2012
Serena Lattante Amelia Conte Marcella Zollino Marco Luigetti Alessandra Del Grande Giuseppe Marangi Angela Romano Alessandro Marcaccio Emiliana Meleo Giulia Bisogni Paolo Maria Rossini Mario Sabatelli

OBJECTIVES To quantify the overall contribution of mutations in the currently known amyotrophic lateral sclerosis (ALS) genes in a large cohort of sporadic patients and to make genotype-phenotype correlations. METHODS Screening for SOD1, TARDBP, FUS, ANG, ATXN2, OPTN, and C9ORF72 was carried out in 480 consecutive patients with sporadic ALS (SALS) and in 48 familial ALS (FALS) index patients ...

Journal: :Rinsho shinkeigaku = Clinical neurology 2017
Syuichi Tetsuka

The advent of next-generation sequencing technology is expected to accelerate the identification of novel genes, and this technology will likely supersede Sanger sequencing. Thus, genome-wide association studies (GWASs) are performed more routinely in an effort to identify disease-susceptibility genes for sporadic amyotrophic lateral sclerosis (ALS). Previously, a Japanese team conducted a larg...

2015
Melissa S. Rotunno Daryl A. Bosco Catherine Ward Reddy Sama Laura Kaushansky

Amyotrophic lateral sclerosis (ALS) is a neurodegenerative disease characterized by a loss of voluntary movement over time, leading to paralysis and death. While 10% of ALS cases are inherited or familial (FALS), the majority of cases (90%) are sporadic (SALS) with unknown etiology. Approximately 20% of FALS cases are genetically linked to a mutation in the anti-oxidizing enzyme, superoxide dis...

Journal: :Brain : a journal of neurology 2010
Adam K Walker Manal A Farg Chris R Bye Catriona A McLean Malcolm K Horne Julie D Atkin

Amyotrophic lateral sclerosis is a rapidly progressing fatal neurodegenerative disease characterized by the presence of protein inclusions within affected motor neurons. Endoplasmic reticulum stress leading to apoptosis was recently recognized to be an important process in the pathogenesis of sporadic human amyotrophic lateral sclerosis as well as in transgenic models of mutant superoxide dismu...

Journal: :Amyotrophic lateral sclerosis & frontotemporal degeneration 2013
John Ravits Stanley Appel Robert H Baloh Richard Barohn Benjamin Rix Brooks Lauren Elman Mary Kay Floeter Christopher Henderson Catherine Lomen-Hoerth Jeffrey D Macklis Leo McCluskey Hiroshi Mitsumoto Serge Przedborski Jeffrey Rothstein John Q Trojanowski Leonard H van den Berg Steven Ringel

Amyotrophic lateral sclerosis (ALS) is characterized phenotypically by progressive weakness and neuropathologically by loss of motor neurons. Phenotypically, there is marked heterogeneity. Typical ALS has mixed upper motor neuron (UMN) and lower motor neuron (LMN) involvement. Primary lateral sclerosis has predominant UMN involvement. Progressive muscular atrophy has predominant LMN involvement...

نمودار تعداد نتایج جستجو در هر سال

با کلیک روی نمودار نتایج را به سال انتشار فیلتر کنید