نتایج جستجو برای: transcript accumulation

تعداد نتایج: 178171  

2012
Olivier Cotsaftis Darren Plett Neil Shirley Mark Tester Maria Hrmova

The HKT family of Na(+) and Na(+)/K(+) transporters is implicated in plant salinity tolerance. Amongst these transporters, the cereal HKT1;4 and HKT1;5 are responsible for Na(+) exclusion from photosynthetic tissues, a key mechanism for plant salinity tolerance. It has been suggested that Na(+) is retrieved from the xylem transpiration stream either in the root or the leaf sheath, protecting th...

2002
Sven Danckwardt Gabriele Neu-Yilik Rolf Thermann Ute Frede Matthias W. Hentze Andreas E. Kulozik

Nonsense-mediated mRNA decay (NMD) represents a phylogenetically widely conserved splicingand translation-dependent mechanism that eliminates transcripts with premature translation stop codons and suppresses the accumulation of C-terminally truncated peptides. Elimination of frameshifted transcripts that result from faulty splicing may be an important function of NMD. To test this hypothesis di...

Journal: :Plant physiology 2001
R A Winz I T Baldwin

Attack by the specialist herbivore, Manduca sexta, on its native host Nicotiana attenuata Torr. ex Wats. produces a dramatic ethylene release, a jasmonate burst, and a suppression of the nicotine accumulation that results from careful simulations of the herbivore's damage. Methyl-jasmonate (MeJA) treatment induces nicotine biosynthesis. However, this induction can be suppressed by ethylene as p...

Journal: :Blood 2002
Sven Danckwardt Gabriele Neu-Yilik Rolf Thermann Ute Frede Matthias W Hentze Andreas E Kulozik

Nonsense-mediated mRNA decay (NMD) represents a phylogenetically widely conserved splicing- and translation-dependent mechanism that eliminates transcripts with premature translation stop codons and suppresses the accumulation of C-terminally truncated peptides. Elimination of frameshifted transcripts that result from faulty splicing may be an important function of NMD. To test this hypothesis ...

Journal: :The Plant cell 1998
Chivasa Carr

Salicylhydroxamic acid (SHAM), an inhibitor of alternative oxidase (AOX), blocks salicylic acid-induced resistance to tobacco mosaic virus (TMV) but does not inhibit pathogenesis-related PR-1 protein synthesis or resistance to fungal and bacterial pathogens. We found that the synthetic resistance-inducing chemical 2, 6-dichloroisonicotinic acid also induced Aox transcript accumulation and SHAM-...

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