نتایج جستجو برای: ژن p15ink4b

تعداد نتایج: 15970  

Journal: :Acta Crystallographica Section A Foundations and Advances 2017

Journal: :International journal of oncology 2007
Yuyang Christine Cai Chi K So Alex Yan Nie Yunlong Song Guang-Yu Yang Li-Dong Wang Xin Zhao Terri Goss Kinzy Chung S Yang

In order to identify representative genetic alterations in esophageal squamous cell carcinomas (ESCC) and useful markers for future early detection, 34 ESCC samples with neighboring normal epithelia and 30 esophageal biopsy samples from Linzhou, P.R. China, were studied. Of the 38 microsatellite markers selected, half were linked with tumor suppressors. More than 40% of the tumor samples showed...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2000
M Seike A Gemma Y Hosoya S Hemmi Y Taniguchi Y Fukuda N Yamanaka S Kudoh

The p16INK4 gene, which is a tumor suppressor gene, is frequently altered in lung cancers. Hypermethylation of the promoter region of the p16INK4 gene seems to be the major mechanism through which p16INK4 become inactivated. Hypermethylation of the p16INK4 gene was reported to occur at an early stage in lung cancer. To determine whether the change in p16INK4 methylation status occurs at the lat...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2006
Roger R Gomis Claudio Alarcón Wei He Qiongqing Wang Joan Seoane Alex Lash Joan Massagué

Transforming growth factor beta (TGF-beta) signals through activation of Smad transcription factors. Activated Smad proteins associate with different DNA-binding cofactors for the recognition and regulation of specific target genes. Members of the forkhead box O family (FoxO1, FoxO3, and FoxO4) play such a role in the induction of the cyclin-dependent kinase inhibitors p15Ink4b and p21Cip1. To ...

Journal: :Genetics and molecular research : GMR 2008
L O Almeida A C Custódio J J Araújo J A Rey J R W Almeida M J Santos C A Clara C Casartelli

Cancer is one of the most common and severe problems in clinical medicine, and nervous system tumors represent about 2% of the types of cancer. The central role of the nervous system in the maintenance of vital activities and the functional consequences of the loss of neurons can explain how severe brain cancers are. The cell cycle is a highly complex process, with a wide number of regulatory p...

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