نتایج جستجو برای: acetaminophen toxicity

تعداد نتایج: 127527  

Journal: :Toxicological sciences : an official journal of the Society of Toxicology 2009
Alison H Harrill Pamela K Ross Daniel M Gatti David W Threadgill Ivan Rusyn

Toxicogenomic studies are increasingly used to uncover potential biomarkers of adverse health events, enrich chemical risk assessment, and to facilitate proper identification and treatment of persons susceptible to toxicity. Current approaches to biomarker discovery through gene expression profiling usually utilize a single or few strains of rodents, limiting the ability to detect biomarkers th...

Journal: :Genome research 2010
Hong-Hsing Liu Peng Lu Yingying Guo Erin Farrell Xun Zhang Ming Zheng Betty Bosano Zhaomei Zhang John Allard Guochun Liao Siyu Fu Jinzhi Chen Kimberly Dolim Ayako Kuroda Jonathan Usuka Janet Cheng William Tao Kevin Welch Yanzhou Liu Joseph Pease Steve A de Keczer Mohammad Masjedizadeh Jing-Shan Hu Paul Weller Tim Garrow Gary Peltz

Acetaminophen-induced liver toxicity is the most frequent precipitating cause of acute liver failure and liver transplant, but contemporary medical practice has mainly focused on patient management after a liver injury has been induced. An integrative genetic, transcriptional, and two-dimensional NMR-based metabolomic analysis performed using multiple inbred mouse strains, along with knowledge-...

Journal: :Toxicological sciences : an official journal of the Society of Toxicology 2003
Janet Dowdy Stacey Brower Michael R Miller

The purpose of this study was to test the hypothesis that acetaminophen would alter an estrogen-regulated process in human cells that express endogenous estrogen receptor alpha and beta (ERalpha and ERbeta). Specifically, the extent to which acetaminophen altered the expression of estrogen-inducible alkaline phosphatase in endometrial adenocarcinoma (Ishikawa) cells and directly interacted with...

2015
Attalla Farag El-Kott Mashael Mohammed Bin-Meferij

BACKGROUND Severe destructive hepatic injuries can be induced by acetaminophen overdose and may lead to acute hepatic failure. OBJECTIVE To investigate the ameliorative effects of Arctium lappa root extract on acetaminophen-induced hepatotoxicity. METHODS Rats were divided into 4 groups: normal control group, Arctium lappa extract group, acetaminophen-injected group, and acetaminophen treat...

ژورنال: طلوع بهداشت یزد 2016
احرامپوش, محمد حسن, حسینی, هیوا, ملکوتیان, محمد, پورشعبان مازندرانی, ملیحه,

Introduction: Pharmaceutical wastes are in group of emerging contaminants. These materials cannot be removed completely with conventional treatment methods of water and wastewater. Drug and its metabolites, due to the potential risks toxic and toxicity potential to humans and environment should be removed from the water resources. In this study, Acetaminophen photo catalytic removal from aqueou...

2015
Mohsen Ebrahimi Seyed Reza Mousavi Alireza Ghassemi Toussi Hamidreza Reihani Farhad Bagherian

BACKGROUND N-acetylcysteine (NAC) has been used as a classic treatment for hepatotoxicity induced by N-acetyl-p-benzoquinone imine (NAPQI) as a metabolite of acetaminophen. However, cimetidine theoretically can reduce the production of toxic metabolites through the inhibition of cytochrome p450, and it recently was proposed as a complementary treatment for acetaminophen toxicity. OBJECTIVE Th...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2014
Ehsan Kheradpezhouh Linlin Ma Arthur Morphett Greg J Barritt Grigori Y Rychkov

Acetaminophen (paracetamol) is the most frequently used analgesic and antipyretic drug available over the counter. At the same time, acetaminophen overdose is the most common cause of acute liver failure and the leading cause of chronic liver damage requiring liver transplantation in developed countries. Acetaminophen overdose causes a multitude of interrelated biochemical reactions in hepatocy...

Journal: :The Journal of pharmacology and experimental therapeutics 1983
D R Abernethy D J Greenblatt M Divoll B Ameer R I Shader

Fourteen healthy volunteers received a single i.v. dose of antipyrine (1.2 g) on two occasions, once before and once during cimetidine treatment (300 mg every 6 hr). In a similar manner, 8 subjects received diazepam (10 mg), 11 subjects received acetaminophen (650 mg) and 8 subjects received lorazepam (2 mg), all by the i.v. route, once before and once during cimetidine coadministration. Pharma...

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