نتایج جستجو برای: ada2

تعداد نتایج: 189  

2011
Milena Novakova Tomas Dolezal

Extra-cellular adenosine is an important regulator of inflammatory responses. It is generated from released ATP by a cascade of ectoenzymes and degraded by adenosine deaminase (ADA). There are two types of enzymes with ADA activity: ADA1 and ADGF/ADA2. ADA2 activity originates from macrophages and dendritic cells and is associated with inflammatory responses in humans and rats. Drosophila posse...

Journal: :Clinical Chemistry and Laboratory Medicine 2021

Abstract Objectives The aim of the present study was to validate a commercially available automated assay for measurement total adenosine deaminase (tADA) and its isoenzymes (ADA1 ADA2) in saliva fast accurate way, evaluate possible changes these analytes individuals with SARS-CoV-2 infection. Methods validation, addition evaluation precision accuracy, included analysis effects main procedures ...

2015
F Schena S Volpi R Caorsi C Pastorino F Penco F Kalli A Omenetti S Chiesa A Bertoni P Picco G Filaci I Aksentijevich A Grossi I Ceccherini A Martini E Traggiai M Gattorno

Introduction ADA2 deficiency, a recently described disease, is characterized by systemic vasculopathy and episodes of strokes. The defect is due to a loss of function mutation of CECR1 gene, codifying for Adenosine Deaminase 2 protein. This protein regulates the catabolism of extracellular adenosine, which we have recently shown is an important regulator of Class Switch Recombination in B lymph...

2013
E Levy-Lahad P Elkan-Navon R Segel SB Pierce T Walsh J Barash S Padeh A Zlotogorski YY Berkun JJ Press M Mukamel PJ Hashkes LL Harel M Tekin F Yalcinkaya O Kasapcopur EF Emirogullari MK Lee RE Klevit PF Renbaum A Weinberg-Shukron S Zeligson D Marek-Yagel M Shohat A Singer E Pras AA Rubinow Y Anikster M-C King

OR13-002 Recessive mutations in CECR1, encoding adenosine deaminase 2 (ADA2), cause systemic and cutaneous polyarteritis nodosa (PAN) E Levy-Lahad, P Elkan-Navon, R Segel, SB Pierce, T Walsh, J Barash, S Padeh, A Zlotogorski, YY Berkun, JJ Press, M Mukamel, PJ Hashkes, LL Harel, M Tekin, F Yalcinkaya, O Kasapcopur, EF Emirogullari, MK Lee, RE Klevit, PF Renbaum, A Weinberg-Shukron, S Zeligson, ...

2015
MS Severino R Caorsi C Gandolfo C Martinetti A Martini M Gattorno

Methods We reviewed the contrast-enhanced brain MR, MR angiography (MRA), and digital subtraction angiography (DSA) examinations of 3 male patients with a confirmed molecular diagnosis of ADA2 deficiency and CNS involvement: two brothers (R312X and E328D mutations in compound heterozygosis) and a third unrelated patient (T360A homozygosis). Age at first MR examination was 6 years, 1 year 5 mont...

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