نتایج جستجو برای: bad gene

تعداد نتایج: 1170207  

2012
Geoff Davies

A profession that claims to understand economies, and that has gained power over the greater part of our societies, has big responsibilities. The fundamental responsibility is to ensure its perception of economies gives some useful guidance to the behaviour of real economies. Here mainstream economics fails utterly, and has been failing for a long time. Worse, it actively resists alternative vi...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2011
Douglas C Marchion Hope M Cottrill Yin Xiong Ning Chen Elona Bicaku William J Fulp Nisha Bansal Hye Sook Chon Xiaomang B Stickles Siddharth G Kamath Ardeshir Hakam Lihua Li Dan Su Carolina Moreno Patricia L Judson Andrew Berchuck Robert M Wenham Sachin M Apte Jesus Gonzalez-Bosquet Gregory C Bloom Steven A Eschrich Said Sebti Dung-Tsa Chen Johnathan M Lancaster

PURPOSE Despite initial sensitivity to chemotherapy, ovarian cancers (OVCA) often develop drug resistance, which limits patient survival. Using specimens and/or genomic data from 289 patients and a panel of cancer cell lines, we explored genome-wide expression changes that underlie the evolution of OVCA chemoresistance and characterized the BCL2 antagonist of cell death (BAD) apoptosis pathway ...

Journal: :The Journal of Cell Biology 2003
William A. Wells

utrition and cell death are integrated in a single mitochondrial complex, according to Nika Danial, Stanley Korsmeyer (Harvard Medical School, Boston, MA), and colleagues. The complex was discovered after gel filtration of the pro-apoptotic BAD protein, and contains BAD, a phosphatase, kinase, and kinase-anchoring protein directed at BAD, and glucokinase. In cells lacking BAD, the complex falls...

Journal: :The Journal of biological chemistry 1997
S Ottilie J L Diaz W Horne J Chang Y Wang G Wilson S Chang S Weeks L C Fritz T Oltersdorf

Bad, an inducer of programmed cell death, was recently isolated from a mouse cDNA library by its ability to bind to the anti-apoptotic protein BCL-2. Sequence analysis suggested that Bad was a member of the BCL-2 gene family that encodes both inducers and inhibitors of programmed cell death. To further analyze the role of BAD in the network of homo- and heterodimers formed by the BCL-2 family, ...

Journal: :Cancer research 2010
Kuntebommanahalli N Thimmaiah John B Easton Peter J Houghton

Rapamycin-induced apoptosis in sarcoma cells is inhibited by insulin-like growth factor-I (IGF-I) through a signaling pathway independent of Ras-extracellular signal-regulated kinase 1/2 and Akt. IGF-I induces Bad phosphorylation (Ser112, Ser136, and Ser155) in a pathway involving phosphoinositide 3' kinase (PI3K) and protein kinase C (PKC; mu, epsilon, or theta) resulting in sequestering Bad f...

Journal: :The Journal of Cell Biology 2003
William A. Wells

utrition and cell death are integrated in a single mitochondrial complex, according to Nika Danial, Stanley Korsmeyer (Harvard Medical School, Boston, MA), and colleagues. The complex was discovered after gel filtration of the pro-apoptotic BAD protein, and contains BAD, a phosphatase, kinase, and kinase-anchoring protein directed at BAD, and glucokinase. In cells lacking BAD, the complex falls...

2011
Douglas C. Marchion Hope M. Cottrill Yin Xiong Ning Chen Elona Bicaku William J. Fulp Nisha Bansal Hye Sook Chon Xiaomang B. Stickles Siddharth G. Kamath Ardeshir Hakam Lihua Li Dan Su Carolina Moreno Patricia L. Judson Andrew Berchuck Robert M. Wenham Sachin M. Apte Jesus Gonzalez-Bosquet Gregory C. Bloom Steven A. Eschrich Said Sebti Johnathan M. Lancaster

Purpose: Despite initial sensitivity to chemotherapy, ovarian cancers (OVCA) often develop drug resistance, which limits patient survival. Using specimens and/or genomic data from 289 patients and a panel of cancer cell lines, we explored genome-wide expression changes that underlie the evolution of OVCA chemoresistance and characterized the BCL2 antagonist of cell death (BAD) apoptosis pathway...

Journal: :The Journal of neuroscience : the official journal of the Society for Neuroscience 2000
J E Springer R D Azbill S A Nottingham S E Kennedy

We report here that activation of the caspase-3 apoptotic cascade in spinal cord injury is regulated, in part, by calcineurin-mediated BAD dephosphorylation. BAD, a proapoptotic member of the bcl-2 gene family, is rapidly dephosphorylated after injury, dissociates from 14-3-3 in the cytosol, and translocates to the mitochondria of neurons where it binds to Bcl-x(L). Pretreatment of animals with...

Journal: :Journal of the American College of Cardiology 2019

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