نتایج جستجو برای: cd74

تعداد نتایج: 788  

Journal: :The FASEB Journal 2021

Constitutively expressed by innate immune cells, the cytokine macrophage migration inhibitory factor (MIF) initiates host responses and drives pathogenic in infectious, inflammatory, autoimmune diseases. Dendritic cells (DCs) express high levels of MIF, but role MIF DC function remains poorly characterized. As is critical for surveillance, we investigated whether promoted DCs. In classical tran...

2007
Frida Lantner Diana Starlets Yael Gore Liat Flaishon Ayala Yamit-Hezi Rivka Dikstein Lin Leng Richard Bucala Yossy Machluf Moshe Oren Idit Shachar

Most mature follicular B cells circulate within the periphery in a quiescent state, without actively contributing to an acute immune response. Lasting B-cell persistence in the periphery is dependent on survival signals that are transduced by cell surface receptors. We recently demonstrated that cell surface CD74 controls mature B-cell survival. Stimulation of cell surface CD74 leads to NFB act...

Journal: :The Journal of Experimental Medicine 2003
Lin Leng Christine N. Metz Yan Fang Jing Xu Seamas Donnelly John Baugh Thomas Delohery Yibang Chen Robert A. Mitchell Richard Bucala

Macrophage migration inhibitory factor (MIF) accounts for one of the first cytokine activities to have been described, and it has emerged recently to be an important regulator of innate and adaptive immunity. MIF is an upstream activator of monocytes/macrophages, and it is centrally involved in the pathogenesis of septic shock, arthritis, and other inflammatory conditions. The protein is encode...

Journal: :Molecular and cellular biology 2014
Janna Schneppenheim Susann Hüttl Torben Mentrup Renate Lüllmann-Rauch Michelle Rothaug Michael Engelke Kai Dittmann Ralf Dressel Masatake Araki Kimi Araki Jürgen Wienands Regina Fluhrer Paul Saftig Bernd Schröder

We reported recently that the presenilin homologue signal peptide peptidase-like 2a (SPPL2a) is essential for B cell development by cleaving the N-terminal fragment (NTF) of the invariant chain (li, CD74). Based on this, we suggested that pharmacological modulation of SPPL2a may represent a novel approach to deplete B cells in autoimmune disorders. With regard to reported overlapping substrate ...

2017
In Koo Hwang Joon Ha Park Tae-Kyeong Lee Dae Won Kim Ki-Yeon Yoo Ji Hyeon Ahn Yang Hee Kim Jun Hwi Cho Young-Myeong Kim Moo-Ho Won Seung Myung Moon

Activated M1 microglia secrete proinflammatory cytokines into damaged brain areas. The present study examined activated M1 microglial morphology and expression in the hippocampal Cornu Ammonis (CA) 1 region, which is vulnerable to transient ischemia. Transient cerebral ischemia was performed for 5 min in gerbils, and neuronal death in the CA1 region following transient cerebral ischemia was con...

Journal: :European journal of cell biology 2012
Verena Schwartz Alexander Krüttgen Joachim Weis Christian Weber Tammo Ostendorf Hongqi Lue Jürgen Bernhagen

Macrophage migration inhibitory factor (MIF) is a pleiotropic cytokine that plays a role in innate and adaptive immunity. Depending on the cellular context and disease state, MIF signaling is mediated by its receptors CXCR2, CXCR4 and/or CD74. Although it is known that MIF is endocytosed, the exact mechanism has remained unknown. In exploring the mechanism of MIF endocytosis with biologically a...

Journal: :Journal of immunology 2010
Inbal Binsky Frida Lantner Valentin Grabovsky Nurit Harpaz Lev Shvidel Alain Berrebi David M Goldenberg Lin Leng Richard Bucala Ronen Alon Michal Haran Idit Shachar

The hallmark of chronic lymphocytic leukemia (CLL) is the relentless accumulation of mature lymphocytes, mostly due to their decreased apoptosis. CD74 was recently shown to serve as a survival receptor on CLL cells. In this study, we show that stimulation of CD74 with its natural ligand, migration inhibitory factor, initiates a signaling cascade that results in upregulation of TAp63, which dire...

Journal: :Cancer discovery 2014
Lynnette Fernandez-Cuesta Dennis Plenker Hirotaka Osada Ruping Sun Roopika Menon Frauke Leenders Sandra Ortiz-Cuaran Martin Peifer Marc Bos Juliane Daßler Florian Malchers Jakob Schöttle Wenzel Vogel Ilona Dahmen Mirjam Koker Roland T Ullrich Gavin M Wright Prudence A Russell Zoe Wainer Benjamin Solomon Elisabeth Brambilla Hélène Nagy-Mignotte Denis Moro-Sibilot Christian G Brambilla Sylvie Lantuejoul Janine Altmüller Christian Becker Peter Nürnberg Johannes M Heuckmann Erich Stoelben Iver Petersen Joachim H Clement Jörg Sänger Lucia A Muscarella Annamaria la Torre Vito M Fazio Idoya Lahortiga Timothy Perera Souichi Ogata Marc Parade Dirk Brehmer Martin Vingron Lukas C Heukamp Reinhard Buettner Thomas Zander Jürgen Wolf Sven Perner Sascha Ansén Stefan A Haas Yasushi Yatabe Roman K Thomas

UNLABELLED We discovered a novel somatic gene fusion, CD74-NRG1, by transcriptome sequencing of 25 lung adenocarcinomas of never smokers. By screening 102 lung adenocarcinomas negative for known oncogenic alterations, we found four additional fusion-positive tumors, all of which were of the invasive mucinous subtype. Mechanistically, CD74-NRG1 leads to extracellular expression of the EGF-like d...

2017
Hannelie Korf Laura Breser Jelter Van Hoeck Janet Godoy Dana P Cook Benoit Stijlemans Elien De Smidt Carolien Moyson João Paulo Monteiro Carvalho Mori Cunha Virginia Rivero Conny Gysemans Chantal Mathieu

Macrophages contribute in the initiation and progression of insulitis during type 1 diabetes (T1D). However, the mechanisms governing their recruitment into the islets as well as the manner of retention and activation are incompletely understood. Here, we investigated a role for macrophage migration inhibitory factor (MIF) and its transmembrane receptor, CD74, in the progression of T1D. Our dat...

Journal: :Atlas of Genetics and Cytogenetics in Oncology and Haematology 2014

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