نتایج جستجو برای: glycolysis
تعداد نتایج: 10426 فیلتر نتایج به سال:
Substrate use switches from fatty acids toward glucose in pressure overload-induced cardiac hypertrophy with an acceleration of glycolysis being characteristic. The activation of AMP-activated protein kinase (AMPK) observed in hypertrophied hearts provides one potential mechanism for the acceleration of glycolysis. Here, we directly tested the hypothesis that AMPK causes the acceleration of gly...
It is now well known that tumor tissues, either in the form of solid tumors or as ascites cells, have a high rate of aerobic and anaerobic glycolysis. A theory on the origin of the tumor cell as proposed by Warburg (1,2) states that a normal cell becomes malignant as the result of impairment to its respiratory processes. Warburg has stated (1, 2) that in order for these cells to survive they mu...
BACKGROUND Cancer cells show increased glycolysis and take advantage of this metabolic pathway to generate ATP. The TP53-induced glycolysis and apoptosis regulator (TIGAR) inhibits aerobic glycolysis and protects tumor cells from intracellular reactive oxygen species (ROS)-associated apoptosis. However, the function of TIGAR in glycolysis and survival of acute myeloid leukemia cells remains unc...
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