نتایج جستجو برای: severe combined immune deficiency

تعداد نتایج: 1039889  

Journal: :Anales de pediatria 2014
L Alsina P Llobet-Agulló P Soler-Palacín

a Sección de Alergia e Inmunología Clínica, Hospital Sant Joan de Déu, Barcelona, Spain b Consulta Immunologia i Al·lèrgia Pediàtrica, Hospital General de Granollers, Barcelona, Spain c Unitat de Patologia Infecciosa i Immunodeficiències de Pediatria, Hospital Universitari Vall d’Hebron, Institut de Recerca Vall d’Hebron, Universitat Autònoma de Barcelona, Barcelona, Spain d Grup d’Immunodefici...

فاطمه محجوب, , محمود حقیقت‌نژاد, , مسعود موحدی, ,

Background: Immune deficiency is one of the major causes of morbidity and mortality in the modern world. Primary immunodeficiency comprises a wide range of disorders that mainly manifest in early childhood as devastating infections with opportunistic organisms. Thymic aplasia is found on autopsy of some patients afflicted with immune deficiency disorders, such as DiGeorge syndrome and severe co...

Journal: :iranian journal of immunology 0
soheyla alyasin clinical immunology and allergy, allergy research center farhad abolnezhadian department of pediatrics, division of immunology and allergy, namazi hospital, shiraz university of medical sciences, shiraz maryam khoshkhui department of clinical immunology and allergy, mashhad university of medical science , mashhad, iran

major histocompatibility complex (mhc) class ii deficiency is a primary immunodeficiency disease characterized by abnormality of mhc class ii molecules surface expression on peripheral blood lymphocytes and monocytes. clinical manifestations include extreme susceptibility to viral, bacterial, and fungal infections but the immunodeficiency is not as severe as scid (severe combined immunodeficien...

Journal: :American journal of blood research 2013
Patrick L Stevens Nishitha M Reddy

Immune surveillance is a dynamic process that involves an intact immune system to identify and protect the host against tumor development. The increased understanding of the genetics, infections and hematological malignancies in congenital immune deficiency states supports the concept that impaired T cells and Natural-killer/T cells leads to B-cell lymphoma. Furthermore, severe combined immunod...

Journal: :Blood 2015
Donald B Kohn

In this issue of Blood, Touzot et al report that autologous gene therapy/hematopoietic stem cell transplantation (HSCT) for infants with X-linked severe combined immune deficiency (SCID-X1) lacking a matched sibling donor may have better outcomes than haploidentical (haplo) HSCT. Because gene therapy represents an autologous transplant, it obviates immune suppression before and after transplant...

Journal: :The Journal of clinical investigation 2015
Jolan E Walter Lindsey B Rosen Krisztian Csomos Jacob M Rosenberg Divij Mathew Marton Keszei Boglarka Ujhazi Karin Chen Yu Nee Lee Irit Tirosh Kerry Dobbs Waleed Al-Herz Morton J Cowan Jennifer Puck Jack J Bleesing Michael S Grimley Harry Malech Suk See De Ravin Andrew R Gennery Roshini S Abraham Avni Y Joshi Thomas G Boyce Manish J Butte Kari C Nadeau Imelda Balboni Kathleen E Sullivan Javeed Akhter Mehdi Adeli Reem A El-Feky Dalia H El-Ghoneimy Ghassan Dbaibo Rima Wakim Chiara Azzari Paolo Palma Caterina Cancrini Kelly Capuder Antonio Condino-Neto Beatriz T Costa-Carvalho Joao Bosco Oliveira Chaim Roifman David Buchbinder Attila Kumanovics Jose Luis Franco Tim Niehues Catharina Schuetz Taco Kuijpers Christina Yee Janet Chou Michel J Masaad Raif Geha Gulbu Uzel Rebecca Gelman Steven M Holland Mike Recher Paul J Utz Sarah K Browne Luigi D Notarangelo

Patients with mutations of the recombination-activating genes (RAG) present with diverse clinical phenotypes, including severe combined immune deficiency (SCID), autoimmunity, and inflammation. However, the incidence and extent of immune dysregulation in RAG-dependent immunodeficiency have not been studied in detail. Here, we have demonstrated that patients with hypomorphic RAG mutations, espec...

پایان نامه :دانشگاه آزاد اسلامی واحد علوم پزشکی تهران - دانشکده پزشکی 1389

هدف: تعیین فراوانی کمبود ویتامین d درمراجعه کنندگان به درمانگاه غدد بالغین بیمارستان بوعلی تهران طی سال های1388-1387 مواد و روش ها: مطالعه در سال 1387 در درمانگاه غدد بیمارستان بوعلی آغاز شد. جمعیت مورد مطالعه بیمارانی بودند که جهت درمان به درمانگاه غدد مراجعه کرده بودند. اطلاعات جمع آوری شده شامل مشخصات از جمله سن، جنس، قد، وزن و علت مراجعه به درمانگاه ویافته های آزمایشگاهی مانند سطح سرمی (o...

Journal: :Cell 2016
Jonathan Hoggatt

Adenosine deaminase (ADA) deficiency results in the accumulation of toxic metabolites that destroy the immune system, causing severe combined immunodeficiency (ADA-SCID), often referred to as the "bubble boy" disease. Strimvelis is a European Medicines Agency approved gene therapy for ADA-SCID patients without a suitable bone marrow donor.

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