نتایج جستجو برای: st2l

تعداد نتایج: 68  

2009
Kenjiro Seki Matthew L. Steinhauser M.D Vandna Handa Joseph Gannon Richard T. Lee

BackgroundST2 is an interleukin-1 receptor family member with membranebound (ST2L) and soluble (sST2) isoforms, and, sST2 is a biomarker for poor outcome in patients with myocardial infarction (MI). IL-33, the recently-discovered ligand for ST2, activates NF-kB and thus may regulate apoptotic cell death. We tested the hypothesis that IL-33 is cardioprotective after MI through ST2 signaling. Met...

2014
Antoine Thierry Sébastien Giraud Aurélie Robin Anne Barra Franck Bridoux Virginie Ameteau Thierry Hauet Jean-Philippe Girard Guy Touchard Jean-Marc Gombert André Herbelin

The endogenous molecules high mobility group box 1 (HMGB1) and interleukin-33 (IL-33) have been identified as alarmins, capable of mediating danger signals during tissue damage. Here, we address their possible role as innate-immune mediators in ischemia-reperfusion injury (IRI) following human kidney transplantation. We analysed serum and urinary HMGB1 and IL-33 levels, all determined by enzyme...

Journal: :Arteriosclerosis, thrombosis, and vascular biology 2012
Andrea Caporali Marco Meloni Ashley M Miller Klemens Vierlinger Alessandro Cardinali Gaia Spinetti Audrey Nailor Ezio Faglia Sergio Losa Ambra Gotti Orazio Fortunato Tijana Mitic Manuela Hofner Christa Noehammer Paolo Madeddu Costanza Emanueli

OBJECTIVE The p75 neurotrophin receptor (p75(NTR)) contributes to diabetes mellitus-induced defective postischemic neovascularization. The interleukin-33 receptor ST2 is expressed as transmembrane (ST2L) and soluble (sST2) isoforms. Here, we studied the following: (1) the impact of p75(NTR) in the healing of ischemic and diabetic calf wounds; (2) the link between p75(NTR) and ST2; and (3) circu...

Journal: :Journal of immunology 2012
Lars Blom Lars K Poulsen

The IL-1 family members IL-1β, IL-18, and IL-33 are potent cytokines in relationship to amplifying the CD4(+) T cell cytokine production. To evaluate their impact on in vitro-differentiated human Th1 and Th2 cultures, such cultures were established from naive T cells, purified from healthy blood donors, and reactivated in the presence of IL-1β, IL-18, or IL-33. Interestingly, we observe modifyi...

Journal: :Circulation. Heart failure 2009
Kenjiro Seki Shoji Sanada Anastacia Y Kudinova Matthew L Steinhauser Vandna Handa Joseph Gannon Richard T Lee

BACKGROUND ST2 is an interleukin (IL)-1 receptor family member with membrane-bound (ST2L) and soluble (sST2) isoforms, and sST2 is a biomarker for poor outcome in patients with myocardial infarction (MI). IL-33, the recently discovered ligand for ST2, activates nuclear factor kappaB and thus may regulate apoptotic cell death. We tested the hypothesis that IL-33 is cardioprotective after MI thro...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2015
Toru Hashimoto David A Kass

Heart failure is a global health problem, with an estimated 30–50 million patients diagnosed worldwide (1, 2). Therapeutic advances have reduced early mortality; however, morbidity caused by damaged or failing ventricles remains high. Indeed, heart failure is the most common cause for hospitalization in elderly individuals, with recalcitrant 5-y survival rates remaining worse than most cancers ...

2011
Yeon-Sik Hong Su-Jin Moon Young-Bin Joo Chan-Hong Jeon Mi-La Cho Ji Hyeon Ju Hye-Jwa Oh Yu-Jung Heo Sung-Hwan Park Ho-Youn Kim Jun-Ki Min

The interleukin-33 (IL-33)/ST2 pathway has emerged as an intercellular signaling system that participates in antigen-allergen response, autoimmunity and fibrosis. It has been suggested that IL-33/ST2 signaling has been involved in the pathogenesis of rheumatoid arthritis (RA), because IL-33 and its receptor have been specifically mapped to RA synovium. The aim of this study was to determine the...

Journal: :Journal of immunology 2009
Mariola Kurowska-Stolarska Bartosz Stolarski Peter Kewin Grace Murphy Christopher J Corrigan Sun Ying Nick Pitman Ananda Mirchandani Batika Rana Nico van Rooijen Malcolm Shepherd Charlie McSharry Iain B McInnes Damo Xu Foo Y Liew

Alternatively activated macrophages (AAM) play a crucial role in type 2 immunity. Mice deficient in ST2, a receptor for the latest member of the IL-1 family, IL-33, have impaired type 2 immune responses. We therefore reasoned that IL-33/ST2 signaling may be involved in the differentiation and activation of AAM during airway inflammation. We report here that IL-33 changed the quiescent phenotype...

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