نتایج جستجو برای: a3243g

تعداد نتایج: 187  

Journal: :Graefe's Archive for Clinical and Experimental Ophthalmology 2018

Journal: :Genetic Testing and Molecular Biomarkers 2013

ژورنال: :مجله دیابت و متابولیسم ایران 0
باقر لاریجانی bagher larijani endocrinology and metabolism research center, tehran university of medical sciencesمرکز تحقیقات غدد درون ریز و متابولیسم، دانشگاه علوم پزشکی تهران مسعود هوشمند masoud houshmand national institute for genetic engineering and biotechnologyپژوهشگاه ملی مهندسی ژنتیک و زیست فناوری قمر سلطان دراج ghamar soltan dorraj national institute for genetic engineering and biotechnologyپژوهشگاه ملی مهندسی ژنتیک و زیست فناوری فرزانه درویش زاده farzaneh darvishzadeh endocrinology and metabolism research center, tehran university of medical sciencesمرکز تحقیقات غدد درون ریز و متابولیسم، دانشگاه علوم پزشکی تهران

میتوکندری یکی از ارگان های داخل سلولی است که dna مخصوص به خود دارد. تا کنون تعدادی بیماری ناشی از جهش mtdna گزارش شده است. یکی از این جهش ها، جهش a3243g و حذف 5kb می باشد. عواقب این جهش های ژنی طیفی از بیماری ها را بوجود می آورد که یکی از آنها دیابت نوع 2 است. هدف از این مطالعه تعیین جهش a3243gtrnaleu و حذف 5kb در بیماران دیابتی نوع 2 می باشد. روش ها: dna 130 بیمار دیابت نوع 2 که میزان انسولین ...

Journal: :Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association 2008
Marije Löwik Elena Levtchenko Dineke Westra Patricia Groenen Eric Steenbergen Jan Weening Marc Lilien Leo Monnens Lambert van den Heuvel

BACKGROUND Focal segmental glomerulosclerosis (FSGS) is a major cause of steroid-resistant nephrotic syndrome in childhood with a central role for the podocytes in the pathogenesis. Mutated proteins expressed in podocytes cause proteinuria. The role of combined gene defects in the development of FSGS is less clear. METHODS We analysed seven podocyte genes known to cause proteinuria and FSGS i...

Journal: :Diabetes 2004
Camilla Cervin Brita Liljeström Tiinamaija Tuomi Seija Heikkinen Juha S Tapanainen Leif Groop Corrado M Cilio

The aim of this study was characterization of a family carrying two mutations known to cause monogenic forms of diabetes, the M626K mutation in the HNF1alpha gene (MODY3) and the A3243G in mtDNA. Beta-cell function and insulin sensitivity were assessed with the Botnia clamp. Heteroplasmy of the A3243G mutation and variants in type 2 diabetes susceptibility genes were determined, and transcripti...

Journal: :Genetic testing 2005
Helen E White Victoria J Durston Anneke Seller Carl Fratter John F Harvey Nicholas C P Cross

Disease-causing mutations in mitochondrial DNA (mtDNA) are typically heteroplasmic and therefore interpretation of genetic tests for mitochondrial disorders can be problematic. Detection of low level heteroplasmy is technically demanding and it is often difficult to discriminate between the absence of a mutation or the failure of a technique to detect the mutation in a particular tissue. The re...

Journal: :Brain : a journal of neurology 1998
P F Chinnery N Howell R N Lightowlers D M Turnbull

The majority of pathogenic mitochondrial DNA (mtDNA) mutations are heteroplasmic, with both mutant and wild-type alleles present within the same individual. MtDNA is transmitted only from females to their offspring but a single female can bear offspring who harbour different levels of mutant mtDNA and have a variable phenotype. In single families, this complex genetic and phenotypic variability...

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