نتایج جستجو برای: cxcl5

تعداد نتایج: 442  

Journal: :Annals of the Rheumatic Diseases 2023

Background As a novel myeloid/neutrophil-targeting therapy for systemic lupus erythematosus (SLE), the therapeutic efficacy of administering mouse ( m )CXCL5 to lupus-prone mice have been demonstrated by our group recently [1,2] . Objectives The aim this study is examine pre-clinical safety and establish pharmacokinetic (PK) response human h ease commercialization bring discovery from bench bed...

زمینه و هدف: پپتید فعال کننده نوتروفیل ازمنشا سلول‌های اپیتلیال (Epithelial cell-derived neutrophil-activating peptide,ENA-78) کموکینی است که در برخی بیماری ها نقش دارد. نقش ژن CXCL5 در بروز مقامت سلول ها به انسولین نشان داده شده است که با فعال کردن مسیر Jak2/STAT5/SOCS2، سیگنال دهی انسولین را مهار می کند. اخیرا چند ریختی - (156G/C (rs352046 از این ژن در ناحیه پروموتر گزارش شده است که می تواند ...

2012
Morgane Ravon Marco Berrera Martin Ebeling Ulrich Certa

We have analyzed the off-target activity of two siRNAs (F7-1, F7-2) that knock-down human blood coagulation factor 7 mRNA. F7-1 modulates a significant number of non-target transcripts while F7-2 shows high selectivity for the target transcript under various experimental conditions. The 3'-UTRs of all F7-1 off-target genes show statistically significant enrichment of the reverse complement of t...

Journal: :JCI insight 2016
Wenjun Li Hsi-Min Hsiao Ryuji Higashikubo Brian T Saunders Ankit Bharat Daniel R Goldstein Alexander S Krupnick Andrew E Gelman Kory J Lavine Daniel Kreisel

It is well established that maladaptive innate immune responses to sterile tissue injury represent a fundamental mechanism of disease pathogenesis. In the context of cardiac ischemia reperfusion injury, neutrophils enter inflamed heart tissue, where they play an important role in potentiating tissue damage and contributing to contractile dysfunction. The precise mechanisms that govern how neutr...

Journal: :Biomedical papers of the Medical Faculty of the University Palacky, Olomouc, Czechoslovakia 2012
Zdenka Navratilova Jiri Gallo Frantisek Mrazek Martin Petrek

BACKGROUND AND AIMS Prosthetic Joint Infection (PJI) is a serious complication of Total Joint Arthroplasty (TJA). The Th-17 immune response characterised by IL (interleukin)-17A, IL-17F, IL-23, chemotactic cytokines and their receptors, plays a prominent role in the immune response to invading bacteria. In addition, high expression of IL-17A has been reported in PJI. The aim of this study was t...

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