نتایج جستجو برای: d4z4

تعداد نتایج: 154  

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2007
Manjusha Dixit Eugénie Ansseau Alexandra Tassin Sara Winokur Rongye Shi Hong Qian Sébastien Sauvage Christel Mattéotti Anne M van Acker Oberdan Leo Denise Figlewicz Marietta Barro Dalila Laoudj-Chenivesse Alexandra Belayew Frédérique Coppée Yi-Wen Chen

Facioscapulohumeral muscular dystrophy (FSHD) is an autosomal dominant disorder linked to contractions of the D4Z4 repeat array in the subtelomeric region of chromosome 4q. By comparing genome-wide gene expression data from muscle biopsies of patients with FSHD to those of 11 other neuromuscular disorders, paired-like homeodomain transcription factor 1 (PITX1) was found specifically up-regulate...

2017
Eugénie Ansseau Céline Vanderplanck Armelle Wauters Scott Q. Harper Frédérique Coppée Alexandra Belayew

FacioScapuloHumeral muscular Dystrophy (FSHD) is one of the most prevalent hereditary myopathies and is generally characterized by progressive muscle atrophy affecting the face, scapular fixators; upper arms and distal lower legs. The FSHD locus maps to a macrosatellite D4Z4 repeat array on chromosome 4q35. Each D4Z4 unit contains a DUX4 gene; the most distal of which is flanked by a polyadenyl...

2016
Jocelyn O. Eidahl Carlee R. Giesige Jacqueline S. Domire Lindsay M. Wallace Allison M. Fowler Susan M. Guckes Sara E. Garwick-Coppens Paul Labhart Scott Q. Harper

D4Z4 repeats are present in at least 11 different mammalian species, including humans and mice. Each repeat contains an open reading frame encoding a double homeodomain (DUX) family transcription factor. Aberrant expression of the D4Z4 ORF called DUX4 is associated with the pathogenesis of Facioscapulohumeral muscular dystrophy (FSHD). DUX4 is toxic to numerous cell types of different species, ...

2009
Xueqing Xu Koji Tsumagari Janet Sowden Rabi Tawil Alan P. Boyle Lingyun Song Terrence S. Furey Gregory E. Crawford Melanie Ehrlich

A subtelomeric region, 4q35.2, is implicated in facioscapulohumeral muscular dystrophy (FSHD), a dominant disease thought to involve local pathogenic changes in chromatin. FSHD patients have too few copies of a tandem 3.3-kb repeat (D4Z4) at 4q35.2. No phenotype is associated with having few copies of an almost identical repeat at 10q26.3. Standard expression analyses have not given definitive ...

2010
Nausica Arnoult Caroline Schluth-Bolard Anne Letessier Irena Drascovic Rachida Bouarich-Bourimi Judith Campisi Sahn-ho Kim Amina Boussouar Alexandre Ottaviani Frédérique Magdinier Eric Gilson Arturo Londoño-Vallejo

The mechanisms governing telomere replication in humans are still poorly understood. To fill this gap, we investigated the timing of replication of single telomeres in human cells. Using in situ hybridization techniques, we have found that specific telomeres have preferential time windows for replication during the S-phase and that these intervals do not depend upon telomere length and are larg...

2009
Darko Bosnakovski Randy S. Daughters Zhaohui Xu Jonathan M. W. Slack Michael Kyba

Facioscapulohumeral muscular dystrophy (FSHD) is caused by contractions of D4Z4 repeats at 4q35.2 thought to induce misregulation of nearby genes, one of which, DUX4, is actually localized within each repeat. A conserved ORF (mDUX), embedded within D4Z4-like repeats, encoding a double-homeodomain protein, was recently identified on mouse chromosome 10. We show here that high level mDUX expressi...

2011
Céline Vanderplanck Eugénie Ansseau Sébastien Charron Nadia Stricwant Alexandra Tassin Dalila Laoudj-Chenivesse Steve D. Wilton Frédérique Coppée Alexandra Belayew

BACKGROUND Facioscapulohumeral muscular dystrophy (FSHD) is linked to deletions in 4q35 within the D4Z4 repeat array in which we identified the double homeobox 4 (DUX4) gene. We found stable DUX4 mRNAs only derived from the most distal D4Z4 unit and unexpectedly extended to the flanking pLAM region that provided an intron and a polyadenylation signal. DUX4 encodes a transcription factor express...

2012
Giulia Ricci Isabella Scionti Greta Alì Leda Volpi Virna Zampa Marina Fanin Corrado Angelini Luisa Politano Rossella Tupler Gabriele Siciliano

We report the first case of a heterozygous T78M mutation in the caveolin-3 gene (CAV3) associated with rippling muscle disease and proximal myopathy. The patient displayed also bilateral winged scapula with limited abduction of upper arms and marked asymmetric atrophy of leg muscles shown by magnetic resonance imaging. Immunohistochemistry on the patient's muscle biopsy demonstrated a reduction...

نمودار تعداد نتایج جستجو در هر سال

با کلیک روی نمودار نتایج را به سال انتشار فیلتر کنید