نتایج جستجو برای: familial cancer

تعداد نتایج: 958449  

2004
Justo Lorenzo Bermejo Alfonso García Pérez Kari Hemminki

The known breast cancer susceptibility genes only account for 20% to 25% of the excess familial risk of the disease 1. The present study assessed the contribution of BRCA1/2 mutations and CHEK2 variants to the relative risk of breast cancer for women with affected mothers or sisters. The familial relative risks were estimated by Poisson regression based on the Swedish Family-Cancer Database. Th...

Journal: :Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology 2011
Jie Shen Leo Medico Hua Zhao

BACKGROUND Family history is the strongest risk factor for ovarian cancer. Recent evidence suggests that unidentified BRCA1/2 variations or other genetic events may contribute to familial ovarian cancers. Allelic imbalance (AI) of BRCA1/2 expression, a result of a significant decrease in the ratios between the expression from one allele of BRCA1/2 and the other allele, has been observed in brea...

Journal: :Indian journal of cancer 2010
M F Atoum H M Hourani A Shoter S N Al-Raheem T K Al Muhrib

PURPOSE Staging of breast tumor has important implications for treatment and prognosis. This study aims at pinpointing the frequency of each stage among familial and nonfamilial breast cancers. MATERIALS AND METHODS Ninety-nine Jordanian females diagnosed with familial and nonfamilial breast cancer between 2000 and 2002 were enrolled in this study All breast cancer cases were staged according...

Journal: :Human molecular genetics 2011
Liisa M Pelttari Tuomas Heikkinen Deborah Thompson Anne Kallioniemi Johanna Schleutker Kaija Holli Carl Blomqvist Kristiina Aittomäki Ralf Bützow Heli Nevanlinna

A homozygous mutation in the RAD51C gene was recently found to cause Fanconi anemia-like disorder. Furthermore, six heterozygous deleterious RAD51C mutations were detected in German breast and ovarian cancer families. We screened 277 Finnish familial breast or ovarian cancer patients for RAD51C and identified two recurrent deleterious mutations (c.93delG and c.837+1G>A). These mutations were fu...

Journal: :European journal of cancer prevention : the official journal of the European Cancer Prevention Organisation 2007
Mahaut Ripert Florence Menegaux Yves Perel Françoise Méchinaud Emmanuel Plouvier Virginie Gandemer Patrick Lutz Jean-Pierre Vannier Jean-Pierre Lamagnére Geneviève Margueritte Patrick Boutard Alain Robert Corinne Armari-Alla Martine Munzer Frédéric Millot Lionel de Lumley Christian Berthou Xavier Rialland Brigitte Pautard Jacqueline Clavel

A case-control study was conducted to investigate the role of a familial history of cancer in the etiology of childhood acute leukemia. The history of cancer in the relatives of 472 cases was compared with that of 567 population-based controls. Recruitment was frequency matched on age, sex and region. The familial history of cancer in each child's relatives was reported by the mother in respons...

2006
Kari Hemminki Charlotta Granström Jan Sundquist Justo Lorenzo Bermejo

The Swedish Family-Cancer Database has been used for some 10 years in the study of familial risks at all common sites. In the present paper we describe some of the main features of version VII of this Database, assembled in year 2006. This update included all residents in Sweden born or immigrated in 1932 and later (offspring) with their biological parents, a total of 11.5 million individuals. ...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2007
Lauri Aaltonen Louise Johns Heikki Järvinen Jukka-Pekka Mecklin Richard Houlston

PURPOSE There is a paucity of data quantifying the familial risk of colorectal cancer associated with mismatch repair (MMR)-deficient and MMR-stable tumors. To address this, we analyzed a population-based series of 1,042 colorectal cancer probands with verified family histories. EXPERIMENTAL DESIGN Constitutional DNA from probands was systematically screened for MYH variants and those with ca...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2009
Ming You Daolong Wang Pengyuan Liu Haris Vikis Michael James Yan Lu Yian Wang Min Wang Qiong Chen Dongmei Jia Yan Liu Weidong Wen Ping Yang Zhifu Sun Susan M Pinney Wei Zheng Xiao-Ou Shu Jirong Long Yu-Tang Gao Yong-Bing Xiang Wong-Ho Chow Nat Rothman Gloria M Petersen Mariza de Andrade Yanhong Wu Julie M Cunningham Jonathan S Wiest Pamela R Fain Ann G Schwartz Luc Girard Adi Gazdar Colette Gaba Henry Rothschild Diptasri Mandal Teresa Coons Juwon Lee Elena Kupert Daniela Seminara John Minna Joan E Bailey-Wilson Christopher I Amos Marshall W Anderson

PURPOSE We have previously mapped a major susceptibility locus influencing familial lung cancer risk to chromosome 6q23-25. However, the causal gene at this locus remains undetermined. In this study, we further refined this locus to identify a single candidate gene, by fine mapping using microsatellite markers and association studies using high-density single nucleotide polymorphisms (SNP). E...

Journal: :Clinical cancer research : an official journal of the American Association for Cancer Research 2007
Lorenzo Melchor Emiliano Honrado Jia Huang Sara Alvarez Tara L Naylor María J García Ana Osorio David Blesa Michael R Stratton Barbara L Weber Juan C Cigudosa Nazneen Rahman Katherine L Nathanson Javier Benítez

PURPOSE Familial breast cancer represents 5% to 10% of all breast tumors. Mutations in the two known major breast cancer susceptibility genes, BRCA1 and BRCA2, account for a minority of familial breast cancer, whereas families without mutations in these genes (BRCAX group) account for 70% of familial breast cancer cases. EXPERIMENTAL DESIGN To better characterize and define the genomic differ...

نمودار تعداد نتایج جستجو در هر سال

با کلیک روی نمودار نتایج را به سال انتشار فیلتر کنید