نتایج جستجو برای: myocyte enhancer factor 2

تعداد نتایج: 3167311  

Journal: :The Journal of biological chemistry 2011
Jun-Xing Zhao Wan-Fu Yue Mei-Jun Zhu Min Du

AMP-activated protein kinase (AMPK) is a key regulator of energy metabolism; it is inhibited under obese conditions and is activated by exercise and by many anti-diabetic drugs. Emerging evidence also suggests that AMPK regulates cell differentiation, but the underlying mechanisms are unclear. We hypothesized that AMPK regulates cell differentiation via altering β-catenin expression, which invo...

Journal: :Development 2007
Matthew J Potthoff Eric N Olson

The myocyte enhancer factor 2 (MEF2) transcription factor acts as a lynchpin in the transcriptional circuits that control cell differentiation and organogenesis. The spectrum of genes activated by MEF2 in different cell types depends on extracellular signaling and on co-factor interactions that modulate MEF2 activity. Recent studies have revealed MEF2 to form an intimate partnership with class ...

Journal: :The EMBO journal 2012
S K Pallavi Diana M Ho Chindo Hicks Lucio Miele Spyros Artavanis-Tsakonas

Genetic analyses in Drosophila revealed a synergy between Notch and the pleiotropic transcription factor Mef2 (myocyte enhancer factor 2), which profoundly influences proliferation and metastasis. We show that these hyperproliferative and invasive Drosophila phenotypes are attributed to upregulation of eiger, a member of the tumour necrosis factor superfamily of ligands, and the consequent acti...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2007
Sudeshna Fisch Susan Gray Stephane Heymans Saptarsi M Haldar Baiqiu Wang Otmar Pfister Lei Cui Ajay Kumar Zhiyong Lin Sucharita Sen-Banerjee Hiranmoy Das Christine A Petersen Ulrike Mende Barbara A Burleigh Yan Zhu Yigal M Pinto Ronglih Liao Mukesh K Jain

Cardiac hypertrophy is a common response to injury and hemodynamic stress and an important harbinger of heart failure and death. Herein, we identify the Kruppel-like factor 15 (KLF15) as an inhibitor of cardiac hypertrophy. Myocardial expression of KLF15 is reduced in rodent models of hypertrophy and in biopsy samples from patients with pressure-overload induced by chronic valvular aortic steno...

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