نتایج جستجو برای: ژن hdac4

تعداد نتایج: 16335  

2008
Wen-Ling Wang Yi-Chao Lee Wen-Ming Yang Wen-Chang Chang Ju-Ming Wang

CEBPB, one of the CEBP family members, is a crucial regulator of gene expression during innate immunity, inflammatory responses and adipogenesis. In this study, the EGF-induced increase of CEBPB mRNA is shown to be coincident with the decrease of COX-2 mRNA. We identified that all of the individual CEBPB isoforms, LAP1, LAP2 and LIP, attenuate EGF-induced COX-2 promoter activity. Although incre...

Journal: :Journal of biochemistry 2007
Hideto Hoshino Tomonori George Nishino Satoshi Tashiro Masaya Miyazaki Yoshihiro Ohmiya Kazuhiko Igarashi Sueharu Horinouchi Minoru Yoshida

Bach2 is a member of the BTB-basic region leucine zipper factor family and represses transcription activity directed by the TPA response element, the Maf recognition element (MARE) and the antioxidant-responsive element. Recently, it was reported that upon oxidative stress Bach2 forms nuclear foci surrounding the promyelocytic leukaemia (PML) bodies and specifically represses the transcription ...

Journal: :Molecular medicine reports 2015
Chutima Kaewpiboon Ratakorn Srisuttee Waraporn Malilas Jeong Moon Sangtaek Oh Hye Gwang Jeong Randal N Johnston Wanchai Assavalapsakul Young-Hwa Chung

Despite efforts to develop efficient chemotherapeutic drug strategies to treat cancer, acquired drug resistance is a commonly encountered problem. In the present study, to investigate this phenomenon, human A549 lung cancer cells resistant to the topoisomerase inhibitor etoposide (A549RT‑eto) were used and compared with A549 parental cells. A549RT‑eto cells demonstrated increased resistance to ...

Journal: :The Journal of biological chemistry 2005
Audrey H Wang Serge Grégoire Eleni Zika Lin Xiao Cathy S Li Hongwei Li Kenneth L Wright Jenny P Ting Xiang-Jiao Yang

Eighteen human histone deacetylases (HDACs) have been identified, and according to their sequence similarity to yeast homologs, these enzymes are grouped into distinct classes. Within class II, HDAC4, HDAC5, HDAC7, and HDAC9 share similar domain organization both within the N-terminal extension and the C-terminal catalytic domain, thus forming a subclass known as class IIa. These HDACs function...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2007
A Lahm C Paolini M Pallaoro M C Nardi P Jones P Neddermann S Sambucini M J Bottomley P Lo Surdo A Carfí U Koch R De Francesco C Steinkühler P Gallinari

Previous findings have suggested that class IIa histone deacetylases (HDACs) (HDAC4, -5, -7, and -9) are inactive on acetylated substrates, thus differing from class I and IIb enzymes. Here, we present evidence supporting this view and demonstrate that class IIa HDACs are very inefficient enzymes on standard substrates. We identified HDAC inhibitors unable to bind recombinant human HDAC4 while ...

2013
Michal Mielcarek Christian Landles Andreas Weiss Amyaouch Bradaia Tamara Seredenina Linda Inuabasi Georgina F. Osborne Kristian Wadel Chrystelle Touller Rachel Butler Janette Robertson Sophie A. Franklin Donna L. Smith Larry Park Paul A. Marks Erich E. Wanker Eric N. Olson Ruth Luthi-Carter Herman van der Putten Vahri Beaumont Gillian P. Bates

Histone deacetylase (HDAC) 4 is a transcriptional repressor that contains a glutamine-rich domain. We hypothesised that it may be involved in the molecular pathogenesis of Huntington's disease (HD), a protein-folding neurodegenerative disorder caused by an aggregation-prone polyglutamine expansion in the huntingtin protein. We found that HDAC4 associates with huntingtin in a polyglutamine-lengt...

2016
Il-Ho Park Ju-Hyung Kang Jae-Min Shin Heung-Man Lee

BACKGROUND AND OBJECTIVES Tissue remodeling is believed to cause recalcitrant chronic rhinosinusitis (CRS). Epithelial-mesenchymal transition (EMT) is a novel clinical therapeutic target in many chronic airway diseases related with tissue remodeling. The aim of this study was to investigate the effect of trichostatin A (TSA) on transforming growth factor (TGF)-β1-induced EMT in airway epitheliu...

Journal: :International journal of oncology 2015
Lin Zhu Jiaxing Yang Linhong Zhao Xue Yu Lingyao Wang Fei Wang Yong Cai Jingji Jin

Increasing evidence suggests that the alteration of global histone H4K16 acetylation (H4K16ac) may be involved in several types of cancer. It is known that the global histone H4K16ac level in cells is controlled by several enzymes including histone acetyltransferases (HATs) and histone deacetylases (HDACs). We report in detail which particular enzyme is responsible for global reduction of histo...

2011
Euan A. Stronach Albandri Alfraidi Nona Rama Christoph Datler James B. Studd Roshan Agarwal Tankut G. Guney Charlie Gourley Bryan T. Hennessy Gordon B. Mills Antonello Mai Robert Brown Roberto Dina Hani Gabra

Ovarian cancer frequently acquires resistance to platinum chemotherapy, representing a major challenge for improving patient survival. Recent work suggests that resistant clones exist within a larger drug-sensitive cell population prior to chemotherapy, implying that resistance is selected for rather than generated by treatment. We sought to compare clinically derived, intrapatient paired model...

Journal: :The Biochemical journal 2006
Songyan Han Jun Lu Yu Zhang Cao Cheng Liping Han Xiuli Wang Lin Li Chunyan Liu Baiqu Huang

The critical role of IL-5 (interleukin-5) in eosinophilic inflammation implicates it as a therapeutic target for allergic diseases. The aim of the present study was to elucidate the molecular basis for the involvement of reversible histone acetylation in IL-5 transcriptional regulation. We provide evidence that HDAC4 (histone deacetylase 4) and p300, a known HAT (histone acetyltransferase), rev...

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