نتایج جستجو برای: acquired brain injury

تعداد نتایج: 894361  

Journal: :مجله علوم اعصاب شفای خاتم 0
s kovac university hospital münster, münster, germany s williams university college london, london, uk am domijan university college london, london, uk mc walker university college london, london, uk ay abramov university college london, london, uk

seizure activity has been proposed to result in the generation of reactive oxygen species (ros), which then contribute to seizure-induced neuronal damage and eventually cell death. although the mechanisms of seizure-induced ros generation are unclear, mitochondria and cellular calcium overload have been proposed to have a crucial role. we aim to determine the sources of seizure-induced ros and ...

Journal: :مجله علوم اعصاب شفای خاتم 0
nafise bahrami a. islamic azad university, mashhad branch, mashhad, iran b. member of mashhad neuroscience research group of islamic azad university, mashhad branch, mashhad, iran zahra ghane a. islamic azad university, mashhad branch, mashhad, iran b. member of mashhad neuroscience research group of islamic azad university, mashhad branch, mashhad, iran hasan abbassian neuroscience department, mashhad university of medical sciences, mashhad, iran

introduction: there are several different molecular pathways in tissue damage by ischemic brain injury. the use of antioxidants and free radical scavengers are a matter of attention by some researchers. crocus sativus (saffron) is used previously for protective effects in ischemia state by some researchers. to assess pathologic aspects of neuroprotection of saffron in some susceptible brain are...

Journal: :International Journal of Therapy and Rehabilitation 2008

Journal: :Scandinavian Journal of Occupational Therapy 2020

Journal: :Developmental Medicine & Child Neurology 2021

This commentary is on the original article by Keetley et al. pages 824–830 of this issue.

ژورنال: Iranian Biomedical Journal 2015
Abbasloo, Elham, Dehghan, Fatemeh, Khaksari, Mohammad, Shahrokhi, Nader,

Background: In previous studies, the neuroprotective effect of 17&beta-estradiol in diffuse traumatic brain injury has been shown. This study used ICI 182,780, a non-selective estrogen receptor antagonist, to test the hypothesis that the neuroprotective effect of 17&beta-estradiol in traumatic brain injury is mediated by the estrogen receptors. Methods: The ovariectomized rats were divided into...

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