نتایج جستجو برای: amyloid aβ

تعداد نتایج: 40212  

2014
Erin R. Greiner Jeffery W. Kelly Fernando L. Palhano

Amyloid fibrils are associated with many maladies, including Alzheimer's disease (AD). The isolation of amyloids from natural materials is very challenging because the extreme structural stability of amyloid fibrils makes it difficult to apply conventional protein science protocols to their purification. A protocol to isolate and detect amyloids is desired for the diagnosis of amyloid diseases ...

Journal: :Jacobs journal of molecular and translational medicine 2016
Sibaprasad Maity Yuri L Lyubchenko

Self-assembly and aggregation of amyloid peptides, such as Aβ(1-40) and Aβ(1-42), lead to the development of Alzheimer disease and similar neurodegenerative disorders associated with protein aggregation. The structures of large aggregates, specifically fibrils, are well characterized. However, our understanding about the structure of oligomeric forms of amyloids is incomplete and needs to be ex...

Journal: :Alzheimers & Dementia 2023

Background Among the various risk factors for Alzheimer disease (AD), apolipoprotein E (APOE) genotype ε4 is known to be a strong factor. On other hand, ε2 have protective effect. It not yet clear how amyloid positivity according APOE differs by diagnostic criteria. Also, there were no studies showing ethnic difference in effects of on amyloid-beta (Aβ) between American and Asian. The aim this ...

2012
Xiaqin Sun Yan Zhang

This chapter reviews the major hypotheses in Alzheimer’s disease (AD) research with the focus on amyloid hypothsis. Since amyloid hypothesis of AD pathology was proposed, extracellular amyloid β (Aβ) toxicity and its role of inducing synaptic plasticity and memory function has been studying intensively. Accumulating evidence indicates that Aβ also exists inside the neurons in AD. Intracellular ...

Journal: :The Journal of neuroscience : the official journal of the Society for Neuroscience 2015
Yona Levites Brian O'Nuallain Rama Devudu Puligedda Tomas Ondrejcak Sharad P Adekar Cindy Chen Pedro E Cruz Awilda M Rosario Sallie Macy Alexandra J Mably Dominic M Walsh Ruben Vidal Alan Solomon Daniel Brown Michael J Rowan Todd E Golde Scott K Dessain

Alzheimer's disease (AD) and familial Danish dementia (FDD) are degenerative neurological diseases characterized by amyloid pathology. Normal human sera contain IgG antibodies that specifically bind diverse preamyloid and amyloid proteins and have shown therapeutic potential in vitro and in vivo. We cloned one of these antibodies, 3H3, from memory B cells of a healthy individual using a hybrido...

چاوشیان, ورژینه, خلجی, ناصر , سرکیسیان , جون , سرکیسیان , واغیناک,

   Background & Aims: Alzheimer’s disease is the most common form of dementia which it destroyed memory and thinking skills slowly. There is no cure for the disease and it leads to death eventually. Proline rich peptide (PRP -1) is produced from neurosecretory cells of hypothalamus that has large spectrum of biological action on immune and nervous system . The Aβ1-42 is more effective in the de...

2017
Conrad N. Trumbore

Liquid sheared amyloid-β (Aβ) initiates amyloid cascade reactions, producing unstable, potentially toxic oligomers. There is a need for new analytical tools with which to study these oligomers. A very small bore capillary flow system is proposed as a tool for studying the effects of liquid shear in amyloid research. This simple system consists of injecting a short cylindrical liquid sample plug...

Journal: :The Journal of neuroscience : the official journal of the Society for Neuroscience 2014
Fan Liao Yukiko Hori Eloise Hudry Adam Q Bauer Hong Jiang Thomas E Mahan Katheryn B Lefton Tony J Zhang Joshua T Dearborn Jungsu Kim Joseph P Culver Rebecca Betensky David F Wozniak Bradley T Hyman David M Holtzman

Apolipoprotein E (apoE) is the strongest known genetic risk factor for late onset Alzheimer's disease (AD). It influences amyloid-β (Aβ) clearance and aggregation, which likely contributes in large part to its role in AD pathogenesis. We recently found that HJ6.3, a monoclonal antibody against apoE, significantly reduced Aβ plaque load when given to APPswe/PS1ΔE9 (APP/PS1) mice starting before ...

Journal: :The Journal of neuroscience : the official journal of the Society for Neuroscience 2016
Peng Yuan Jaime Grutzendler

UNLABELLED Aberrant neural hyperactivity has been observed in early stages of Alzheimer's disease (AD) and may be a driving force in the progression of amyloid pathology. Evidence for this includes the findings that neural activity may modulate β-amyloid (Aβ) peptide secretion and experimental stimulation of neural activity can increase amyloid deposition. However, whether long-term attenuation...

Journal: :Neurobiology of aging 2012
Ola Philipson Anna Lord Maciej Lalowski Rabah Soliymani Marc Baumann Johan Thyberg Nenad Bogdanovic Tommie Olofsson Lars O Tjernberg Martin Ingelsson Lars Lannfelt Hannu Kalimo Lars N G Nilsson

The Arctic (p. E693G) mutation in the amyloid-β precursor protein (AβPP) facilitates amyloid-β (Aβ) protofibril formation and generates clinical symptoms of Alzheimer's disease (AD). Here, molecular details of Aβ in post mortem brain were investigated with biochemical and morphological techniques. The basic structure of Arctic plaques resembled cotton wool plaques. However, they appeared ring-f...

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