نتایج جستجو برای: chronic energy deficiencies ced

تعداد نتایج: 1176278  

Journal: :Malaysian journal of nutrition 2009
R K Gautam R Thakur

The paper aims to draw out biosocial correlates of nutrition through body mass index (BMI) and chronic energy deficiency (CED). The findings are based on cross-sectional data of 446 women aged 18-60 years from six different endogamous groups of two ecological zones. The mean age of studied women varied from 31 to 36 years. The mean age at menarche was found to be 14.50±1.32 years. Similarly mea...

2016
Ashraful Islam Nurul Islam Premananda Bharati Saw Aik Golam Hossain

BACKGROUND Early childbearing influences women's health. This study aims to examine the effects of socio-demographic factors on nutritional status of early childbearing mothers in Bangladesh based on Body Mass Index (BMI) as the indicator. METHODS Data was extracted from Bangladesh Demographic and Health Survey (BDHS)-2011. The survey was performed on 17,842 married women aged 15-49. We focus...

Journal: :Stroke 2006
Oliver C Singer Marek C Humpich Helmut Laufs Heiner Lanfermann Helmuth Steinmetz Tobias Neumann-Haefelin

BACKGROUND AND PURPOSE The authors studied clinical features and lesion patterns associated with conjugate eye deviation (CED) in acute stroke. METHODS Consecutive patients with supratentorial stroke (<6 hours after symptom onset) were prospectively recruited. CED was classified according to the National Institute of Health Stroke Scale (NIHSS) item "gaze palsy" as being forced, partial, or a...

Journal: :Development 1992
J Yuan H R Horvitz

Mutations in the gene ced-4 block almost all of the programmed cell deaths that normally occur during Caenorhabditis elegans development. We have cloned the ced-4 gene using a ced-4 mutation caused by the insertion of the transposon Tc4. When microinjected into a ced-4 animal, a 4.4 kb DNA fragment derived from the wild-type strain and corresponding to the region of the Tc4 insertion in the mut...

Journal: :Molecular cell 2000
G M Stanfield H R Horvitz

Loss-of-function mutations in the gene ced-8 lead to the late appearance of cell corpses during embryonic development in C. elegans. ced-8 functions downstream of or in parallel to-the regulatory cell death gene ced-9 and may function as a cell death effector downstream of the caspase encoded by the programmed cell death killer gene ced-3. In ced-8 mutants, embryonic programmed cell death proba...

Journal: :International Journal of Public Health Science 2023

Stunting is a sign of persistent malnutrition during critical period child development, especially in the second trimester pregnancy. This study aimed to determine causes stunting infants between 24 and 59 months. The Kamaipura Health Center Sigi Regency, Indonesia location for research. Data was collected from January December 2020 medical record data. population consisted 156 children under a...

2008
Renu Tyagi

Aim of the present study was to assess and compare the nutritional status and body composition variation among institutionalized and non-institutionalized senior citizens. Height, weight, skinfold thicknesses at five different body sites, waist circumference, hip circumference, heart rate and blood pressure were measured in 457 subjects ranging in age from 60-85 years. This includes 175 elderly...

Journal: :Development 1999
D Wu P J Chen S Chen Y Hu G Nuñez R E Ellis

In the nematode Caenorhabditis elegans, CED-4 plays a central role in the regulation of programmed cell death. To identify proteins with essential or pleiotropic activities that might also regulate cell death, we used the yeast two-hybrid system to screen for CED-4-binding proteins. We identified MAC-1, a member of the AAA family of ATPases that is similar to Smallminded of Drosophila. Immunopr...

2010
Mary Grace Tungdim

A prospective case-control study was done on adult TB cases between the age 20-40 years diagnosed and registered in the different TB centers in Manipur, Northeast India. Cases were defined as subjects who were diagnosed as suffering from TB and taking regular medicines. Controls were apparently healthy subjects who had never suffered from tuberculosis as confirmed from doctors. Anthropometric m...

Journal: :Cell 1996
Shai Shaham H.Robert Horvitz

The C. elegans gene ced-4 is essential for programmed cell death. We report that ced-4 encodes two transcripts and that whereas the major transcript can cause programmed cell death, the minor transcript can act oppositely and prevent programmed cell death, thus defining a novel class of cell death inhibitors. That ced-4 has both cell-killing and cell-protective functions is consistent with prev...

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