نتایج جستجو برای: familial polyposis coli

تعداد نتایج: 208445  

2015
Shan-Shan Jiang Jian-Jun Li Yin Li Long-Jun He Qi-Jing Wang De-Sheng Weng Ke Pan Qing Liu Jing-Jing Zhao Qiu-Zhong Pan Xiao-Fei Zhang Yan Tang Chang-Long Chen Hong-Xia Zhang Guo-Liang Xu Yi-Xin Zeng Jian-Chuan Xia

Familial adenomatous polyposis (FAP) is an autosomal dominant disease manifesting as colorectal cancer in middle-aged patients. Mutations of the adenomatous polyposis coli (APC) gene contribute to both FAP and sporadic or familial colorectal carcinogenesis. Here we describe the identification of the causative APC gene defects associated with FAP in a Chinese pedigree. All patients with FAP were...

Journal: :Cancer research 1998
S M Baker A C Harris J L Tsao T J Flath C E Bronner M Gordon D Shibata R M Liskay

Analysis of two human familial cancer syndromes, hereditary nonpolyposis colorectal cancer and familial adenomatous polyposis, indicates that mutations in either one of four DNA mismatch repair gene homologues or the adenomatous polyposis coli (APC) gene, respectively, are important for the development of colorectal cancer. To further investigate the role of DNA mismatch repair in intestinal tu...

2009
Feride Aydin Claus Ferdinand Eisenberger Andreas Raffel Alexander Rehders Stefan Benedikt Hosch Wolfram Trudo Knoefel

Fistulae between an ileal pouch and the vagina are an uncommon complication of ileal pouch-anal anastomosis following proctocolectomy and mucosectomy in patients with familial adenomatous polyposis coli. Several reports describe the successful use of muscle flaps to close recurrent pouch-vaginal-fistulae (PVF). However, series only contain small numbers and an optimal management has not yet bee...

2007
Masanobu Oshima Hiroko Sugiyama Kyoko Kitagawa Makoto Taketo

The APC gene at human chromosome 5q21 is responsible for familial adenomatous polyposis coli. Furthermore, sporadic cancers of not only colon but also other digestive organs often contain mutations in the APC gene. A dominant mouse mutation Min that was generated by chemical mutagenesis and causes polyposis in the digestive tract is in the mouse homologue of the human APC gene. The APC mRNA is ...

2006
Sean M. Baker Allie C. Harris Jen-Lan Tsao Tom J. Flath C. Eric Bronner Melissa Gordon Darryl Shibata Michael Liskay

Analysis of two human familial cancer syndromes, hereditary nonpol yposis colorectal cancer and familial adenomatous polyposis, indicates that mutations in either one of four DNA mismatch repair gene homo logues or the adenomatous polyposis coli (APC) gene, respectively, are important for the development of colorectal cancer. To further investigate the role of DNA mismatch repair in intestinal ...

Journal: :The Turkish journal of gastroenterology : the official journal of Turkish Society of Gastroenterology 2012
Cumhur İbrahim Başsorgun İrem Hicran Özbudak Gülgün Erdoğan Gülsüm Özlem Elpek Okan Erdoğan Tekinalp Gelen

Desmoid tumors are one of the most common extracolonic manifestations of the familial adenomatous polyposis. However, other soft tissue tumors are seen rarely in patients with familial adenomatous polyposis, including gastrointestinal stromal tumor. There is only one case reported in the literature. We describe a 29-year-old female who developed ileal malignant gastrointestinal stromal tumor 15...

2015
Betul Eser Murat Yıldar

Familial adenomatous polyposis (FAP) is an autosomal dominant syndrome leading to colorectal cancer. This disease appears as a result of germline mutation in adenomatous polyposis coli (APC) gene. The aim of the present study is to report the association between two different nucleotide substitutions detected in a family with FAP. In the proband, p.His1172Gln (c.3516delT) was detected in exon 1...

Journal: :The Tohoku Journal of Experimental Medicine 1987

Journal: :Nihon Rinsho Geka Gakkai Zasshi (Journal of Japan Surgical Association) 1998

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