نتایج جستجو برای: giant axonal neuropathy

تعداد نتایج: 119020  

Journal: :The Journal of Cell Biology 1985
M W Klymkowsky D J Plummer

Giant axonal neuropathy (GAN) results from autosomal recessive mutations (gan-) that affect cytoskeletal organization; specifically, intermediate filaments (IFs) are found collapsed into massive bundles in a variety of different cell types. We studied the gan- fibroblast lines WG321 and WG139 derived from different GAN patients. Although previous studies implied that the gan- IF phenotype was c...

Journal: :The Canadian journal of neurological sciences. Le journal canadien des sciences neurologiques 2014
Puneet Jain Suvasini Sharma Mahesh Kamate Virupaxi Hattiholi Bijoy Patra Anita Mahadevan Satinder Aneja

An 8-year-old boy, born to a third-degree consanguineous couple with no adverse perinatal events, presented with complaints of delayed development and gait abnormalities. He had global developmental delay with independent ambulation achieved at two years of age and monosyllables at 2.5 years of age. There was no regression. His hearing and vision were normal. He had had two episodes of generali...

2013
Puneet Opal Robert D. Goldman

Giant axonal neuropathy (GAN)(1) is a rare autosomal recessive neurological disorder caused by mutations in the GAN gene that encodes gigaxonin, a member of the BTB/Kelch family of E3 ligase adaptor proteins.(1) This disease is characterized by the aggregation of Intermediate Filaments (IF)-cytoskeletal elements that play important roles in cell physiology including the regulation of cell shape...

2016
Hiram Larangeira de Almeida Jr. Gilberto Garcias Ricardo Marques e Silva Stela Laner Batista Fernanda Pasetto

Giant axonal neuropathy is a rare autosomal recessive neurodegenerative disease. The condition is characterized by neurons with abnormally large axons due to intracellular filament accumulation. The swollen axons affect both the peripheral and central nervous system. A 6-year old female patient had been referred to a geneticist reporting problems with walking and hypotonia. At the age of 10, sh...

Journal: :Human molecular genetics 2006
Jianqing Ding Elizabeth Allen Wei Wang Angela Valle Chengbiao Wu Timothy Nardine Bianxiao Cui Jing Yi Anne Taylor Noo Li Jeon Steven Chu Yuen So Hannes Vogel Ravi Tolwani William Mobley Yanmin Yang

Mutations in gigaxonin were identified in giant axonal neuropathy (GAN), an autosomal recessive disorder. To understand how disruption of gigaxonin's function leads to neurodegeneration, we ablated the gene expression in mice using traditional gene targeting approach. Progressive neurological phenotypes and pathological lesions that developed in the GAN null mice recapitulate characteristic hum...

Journal: :Journal of neurology, neurosurgery, and psychiatry 2011
Judith Drenthen Nobuhiro Yuki Jan Meulstee Ellen M Maathuis Pieter A van Doorn Gerhard H Visser Joleen H Blok Bart C Jacobs

BACKGROUND In Guillain-Barré syndrome (GBS), the diversity in electrophysiological subtypes is unexplained but may be determined by geographical factors and preceding infections. Acute motor axonal neuropathy (AMAN) is a frequent GBS variant in Japan and one study proposed that in Japan, Campylobacter jejuni infections exclusively elicit AMAN. In The Netherlands C jejuni is the predominant type...

Journal: :Journal of neurology, neurosurgery, and psychiatry 1997
K C Gorson A H Ropper

OBJECTIVE The neuropathy associated with monoclonal gammopathy of undetermined significance (MGUS) is typically a predominantly demyelinating process that may have additional features of axonal degeneration. Sixteen patients with MGUS and a pure or predominantly axonal neuropathy are reported and compared with 20 consecutive patients with demyelinating neuropathy and MGUS who were seen during t...

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