نتایج جستجو برای: mitokatp

تعداد نتایج: 121  

2013
HUI WANG XIANGRONG ZUO QIANG WANG YANZHE YU LIJUN XIE HONG WANG HONGCHENG WU WEIPING XIE

Apoptosis of human pulmonary artery endothelial cells (HPAECs) is the initial step and triggering event for pulmonary hypertension (PH). However, little is known about the actions of nicorandil on HPAECs in vitro. In the present study, we investigated the anti-apoptotic effect of nicorandil on HPAECs exposed to hypoxia, and explored the underlying mechanism(s) of action. Cell viability was asse...

2012
Alice S. Ho Jasma Rucker Anders O. Garlid Ling Chen Agnieszka Sidor Brian O’Rourke

Authors contributed equally to this work.

2014
Prasad V. G. Katakam Angellica O. Gordon Venkata N. L. R. Sure Ibolya Rutkai David W. Busija

Katakam PV, Gordon AO, Sure VN, Rutkai I, Busija DW. Diversity of mitochondria-dependent dilator mechanisms in vascular smooth muscle of cerebral arteries from normal and insulin-resistant rats. Am J Physiol Heart Circ Physiol 307: H493–H503, 2014. First published June 14, 2014; doi:10.1152/ajpheart.00091.2014.—Mitochondrial depolarization following ATP-sensitive potassium (mitoKATP) channel ac...

2017
Kyung Hye Lee So-Ra Lee Haneul Cho Jong Shin Woo Jung Hee Kang Yun-Mi Jeong Xian Wu Cheng Woo-Shik Kim Weon Kim

Soluble guanylate cyclase (sGC) has been suggested as a therapeutic target for cardiac ischemia-reperfusion (IR) injury. Until now, the molecular mechanism of BAY 60-2770, a sGC activator, in cardiac IR injury has not been assessed. To identify the cardioprotective effects of BAY 60-2770 in IR-injured rat hearts, IR injury was established by occlusion of LAD for 40 min and reperfusion for 7 day...

Journal: :American journal of physiology. Heart and circulatory physiology 2007
Philippe Pasdois Casey L Quinlan Abraham Rissa Liliane Tariosse Béatrice Vinassa Alexandre D T Costa Sandrine V Pierre Pierre Dos Santos Keith D Garlid

We showed recently that mitochondrial ATP-dependent K(+) channel (mitoK(ATP)) opening is required for the inotropic response to ouabain. Because mitoK(ATP) opening is also required for most forms of cardioprotection, we investigated whether exposure to ouabain was cardioprotective. We also began to map the signaling pathways linking ouabain binding to Na(+)-K(+)-ATPase with the opening of mitoK...

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