نتایج جستجو برای: neuronal aging

تعداد نتایج: 220967  

2012
Junyang Jung Changhyun Na Youngbuhm Huh

Aging is associated with neuronal loss, gross weight reduction of the brain, and glial proliferation in the cortex, all of which lead to functional changes in the brain. It is known that oxidative stress is a critical factor in the pathogenesis of aging; additionally, growing evidence suggests that excessive nitric oxide (NO) production contributes to the aging process. However, it is still unc...

2015
Jorge Azpurua Benjamin A. Eaton

Two of the most salient phenotypes of aging are cognitive decline and loss of motor function, both of which are controlled by the nervous system. Cognition and muscle contraction require that neuronal synapses develop and maintain proper structure and function. We review the literature on how normal physiological aging disrupts central and peripheral synapse function including the degradation o...

2017
José M. Frade Noelia López-Sánchez

Journal: :Experimental neurology 1989
S C Bondy J Martin L C Halsall M McKee

Changes in membrane integrity have been implicated as a contributing factor in the aging process. We have examined the relative stability of synaptosomal membranes from young and aged mice, in response to chemically induced damage. The basal characteristics of the resting synaptosomes prepared from mice of either age were indistinguishable, as judged by intracellular levels of ionic calcium and...

2014
Christopher Sinadinos Jordi Valles-Ortega Laura Boulan Estel Solsona Maria F Tevy Mercedes Marquez Jordi Duran Carmen Lopez-Iglesias Joaquim Calbó Ester Blasco Marti Pumarola Marco Milán Joan J Guinovart

Glycogen is a branched polymer of glucose and the carbohydrate energy store for animal cells. In the brain, it is essentially found in glial cells, although it is also present in minute amounts in neurons. In humans, loss-of-function mutations in laforin and malin, proteins involved in suppressing glycogen synthesis, induce the presence of high numbers of insoluble polyglucosan bodies in neuron...

2015
Michela Deleidi Madeline Jäggle Graziella Rubino

As we age, the immune system undergoes a process of senescence accompanied by the increased production of proinflammatory cytokines, a chronic subclinical condition named as "inflammaging". Emerging evidence from human and experimental models suggest that immune senescence also affects the central nervous system and promotes neuronal dysfunction, especially within susceptible neuronal populatio...

Journal: :Zeitschrift fur Naturforschung. C, Journal of biosciences 2001
K Schulpis A Doulgeraki S Tsakiris

The process of brain aging is an interaction of age-related losses and compensatory mechanisms. This review is focused on the changes of the synaptic number and structure, their functional implications, regarding neurotransmission, as well as the electrical activity of neuronal circuits. Moreover, the importance of calcium homeostasis is strongly emphasized. It is also suggested that many neuro...

Journal: :Annals of clinical and translational neurology 2016
Erin L Abner Gregory A Jicha Leslie M Shaw John Q Trojanowski Edward J Goetzl

Plasma neuronal exosomal levels of pathogenic Alzheimer's disease (AD) proteins, cellular survival factors, and lysosomal proteins distinguish AD patients from control subjects, but changes in these exosomal proteins associated with normal aging have not been described for cognitively intact subjects. Plasma neuronal exosomal levels of P-T181-tau, P-S396-tau, Aβ 1-42, cathepsin D, repressor ele...

نمودار تعداد نتایج جستجو در هر سال

با کلیک روی نمودار نتایج را به سال انتشار فیلتر کنید