نتایج جستجو برای: ptch1

تعداد نتایج: 516  

Journal: :Proceedings of the National Academy of Sciences of the United States of America 1999
S H Shin P Kogerman E Lindström R Toftgárd L G Biesecker

Truncation mutations of the GLI3 zinc finger transcription factor can cause Greig cephalopolysyndactyly syndrome (GCPS), Pallister-Hall syndrome (PHS), and postaxial polydactyly type A (PAP-A). GLI3 is homologous to Drosophila Cubitus interruptus (Ci), which regulates the patched (ptc), gooseberry (gsb), and decapentaplegic (dpp) genes. Ci is sequestered in the cytoplasm and is subject to postt...

Journal: :Blood 2012
Sarah Decker Katja Zirlik Lauritte Djebatchie David Hartmann Gabriele Ihorst Annette Schmitt-Graeff Dieter Herchenbach Hassan Jumaa Markus Warmuth Hendrik Veelken Christine Dierks

Hedgehog (HH) signaling is activated in various lymphoid malignancies, but conflicting results exist about its role in chronic lymphocytic leukemia (CLL). Here, we demonstrate that the expression of essential HH pathway components like GLI1, PTCH1, and the HH ligands is highly diverse in CLL. A subset of 36.7% of 60 tested CLL samples responded to all 3 SMOOTHENED (SMO) inhibitors, whereas 40% ...

Journal: :Development 2005
Ayanna F Cooper Kuan Ping Yu Martina Brueckner Lisa L Brailey Linda Johnson James M McGrath Allen E Bale

The hedgehog (Hh) pathway is conserved from Drosophila to humans and plays a key role in embryonic development. In addition, activation of the pathway in somatic cells contributes to cancer development in several tissues. Suppressor of fused is a negative regulator of Hh signaling. Targeted disruption of the murine suppressor of fused gene (Sufu) led to a phenotype that included neural tube def...

2014
Frederique Zindy Daisuke Kawauchi Youngsoo Lee Olivier Ayrault Leila Ben Merzoug Peter J. McKinnon Andrea Ventura Martine F. Roussel

The miR-17∼92 cluster family is composed of three members encoding microRNAs that share seed sequences. To assess their role in cerebellar and medulloblastoma (MB) development, we deleted the miR-17∼92 cluster family in Nestin-positive neural progenitors and in mice heterozygous for the Sonic Hedgehog (SHH) receptor Patched 1 (Ptch1(+/-)). We show that mice in which we conditionally deleted the...

Journal: :Journal of oral pathology & medicine : official publication of the International Association of Oral Pathologists and the American Academy of Oral Pathology 2014
Clarissa Araújo Silva Gurgel Marcilei Eliza Cavichiolli Buim Kátia Cândido Carvalho Caroline Brandi Schlaepfer Sales Mitermayer Galvão Reis Renata Oliveira de Souza Ludmila de Faro Valverde Roberto Almeida de Azevedo Jean Nunes Dos Santos Fernando Augusto Soares Eduardo Antônio Gonçalves Ramos

BACKGROUND Sonic hedgehog (SHH) pathway activation has been identified as a key factor in the development of many types of tumors, including odontogenic tumors. Our study examined the expression of genes in the SHH pathway to characterize their roles in the pathogenesis of keratocystic odontogenic tumors (KOT) and ameloblastomas (AB). METHODS We quantified the expression of SHH, SMO, PTCH1, S...

Journal: :Cancer research 2012
Dennis Liang Fei Avencia Sanchez-Mejias Zhiqiang Wang Colin Flaveny Jun Long Samer Singh Jezabel Rodriguez-Blanco Robert Tokhunts Camilla Giambelli Karoline J Briegel Wolfgang A Schulz A Jay Gandolfi Margaret Karagas Teresa A Zimmers Merce Jorda Pablo Bejarano Anthony J Capobianco David J Robbins

The role of Hedgehog (HH) signaling in bladder cancer remains controversial. The gene encoding the HH receptor and negative regulator PATCHED1 (PTCH1) resides on a region of chromosome 9q, one copy of which is frequently lost in bladder cancer. Inconsistent with PTCH1 functioning as a classic tumor suppressor gene, loss-of-function mutations in the remaining copy of PTCH1 are not commonly found...

Journal: :Atlas of Genetics and Cytogenetics in Oncology and Haematology 2010

Journal: :Birth defects research. Part A, Clinical and molecular teratology 2010
Tonia C Carter Anne M Molloy Faith Pangilinan James F Troendle Peadar N Kirke Mary R Conley David J A Orr Michael Earley Eamon McKiernan Ena C Lynn Anne Doyle John M Scott Lawrence C Brody James L Mills

BACKGROUND Suggestive, but not conclusive, studies implicate many genetic variants in oral cleft etiology. We used a large, ethnically homogenous study population to test whether reported associations between nonsyndromic oral clefts and 12 genes (CLPTM1, CRISPLD2, FGFR2, GABRB3, GLI2, IRF6, PTCH1, RARA, RYK, SATB2, SUMO1, TGFA) could be confirmed. METHODS Thirty-one single nucleotide polymor...

2015
Feng Shuang Ying Zhou Shu-Xun Hou Jia-Liang Zhu Yan Liu Chun-Li Zhang Jia-Guang Tang

Indian Hedgehog (HH) has been shown to be involved in osteoarthritis (OA) in articular joints, where there is evidence that Indian HH blockade could ameliorate OA. It seems to play a prominent role in development of the intervertebral disc (IVD) and in postnatal maintenance. There is little work on IHH in the IVD. Hence the aim of the current study was to investigate the role of Indian Hedgehog...

Journal: :The Journal of clinical investigation 2011
Elly Sau-Wai Ngan Maria-Mercè Garcia-Barceló Benjamin Hon-Kei Yip Hiu-Ching Poon Sin-Ting Lau Carmen Ka-Man Kwok Eric Sat Mai-Har Sham Kenneth Kak-Yuen Wong Brandon J Wainwright Stacey S Cherny Chi-Chung Hui Pak Chung Sham Vincent Chi-Hang Lui Paul Kwong-Hang Tam

Hirschsprung (HSCR) disease is a complex genetic disorder attributed to a failure of the enteric neural crest cells (ENCCs) to form ganglia in the hindgut. Hedgehog and Notch are implicated in mediating proliferation and differentiation of ENCCs. Nevertheless, how these signaling molecules may interact to mediate gut colonization by ENCCs and contribute to a primary etiology for HSCR are not kn...

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