نتایج جستجو برای: triplet repeat primed

تعداد نتایج: 96107  

2008
Anna J. Jasinska Piotr Kozlowski Wlodzimierz J. Krzyzosiak

Numerous human transcripts contain tandem repeats of trinucleotide motifs, the function of which remains unknown. In this study we used the available gene expression EST data to characterize the abundance of a large group of these transcripts in different tissues and determine the mRNAs which had the highest contribution to the observed levels of transcripts containing different types of the CN...

2016
Elaine M. Cleary Suvankar Pal Tara Azam David J. Moore Robert Swingler George Gorrie Laura Stephenson Shuna Colville Siddharthan Chandran Mary Porteous Jon P. Warner

Due to the GC-rich, repetitive nature of C9orf72 hexanucleotide repeat expansions, PCR based detection methods are challenging. Several limitations of PCR have been reported and overcoming these could help to define the pathogenic range. There is also a need to develop improved repeat-primed PCR assays which allow detection even in the presence of genomic variation around the repeat region. We ...

2005
Shigeo Okada Riu Yamashita Kengo Kinoshita Kenta Nakai

Journal: :Human molecular genetics 1997
C E Pearson A Ewel S Acharya R A Fishel R R Sinden

The expansion of trinucleotide repeat sequences is associated with several neurodegenerative diseases. The mechanism of this expansion is unknown but may involve slipped-strand structures where adjacent rather than perfect complementary sequences of a trinucleotide repeat become paired. Here, we have studied the interaction of the human mismatch repair protein MSH2 with slipped-strand structure...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2009
Michael Molla Arthur Delcher Shamil Sunyaev Charles Cantor Simon Kasif

Length variation in short tandem repeats (STRs) is an important family of DNA polymorphisms with numerous applications in genetics, medicine, forensics, and evolutionary analysis. Several major diseases have been associated with length variation of trinucleotide (triplet) repeats including Huntington's disease, hereditary ataxias and spinobulbar muscular atrophy. Using the reference human genom...

Journal: :Biochemistry 1996
C E Pearson R R Sinden

Most models proposed to explain the disease-associated expansion of (CTG)n.(CAG)n and (CGG)n.(CCG)n trinucleotide repeats include the formation of slipped strand DNA structures during replication; however, physical evidence for these alternative DNA secondary structures has not been reported. Using cloned fragments from the myotonic dystrophy (DM) and fragile X syndrome (FRAXA) loci containing ...

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